DNA repair gene XPD and susceptibility to arsenic-induced hyperkeratosis

Toxicology Letters

Volumn 143, Issue 2, 2003, Pages 123-131

  Ahsan, Habibul ^a,b   Chen, Yu ^a   Wang, Qiao ^a   Slavkovich, Vesna ^a   Graziano, Joseph H ^a   Santella, Regina M ^a,b  

^a Columbia University ^*  (United States)

^b Och Spine at New York Presbyterian Hospitals   (United States)

Author keywords

Arsenic; DNA repair gene; Gene environmental interaction; Genetic epidemiology; Genetic polymorphism; Genetic susceptibility; Molecular epidemiology; XPD

Indexed keywords

ARSENIC; INORGANIC COMPOUND;

ADULT; AMINO ACID SUBSTITUTION; ARTICLE; BANGLADESH; CASE CONTROL STUDY; CLINICAL ARTICLE; CODON; CONTROLLED STUDY; CREATININE URINE LEVEL; DNA REPAIR; FEMALE; GENE; GENE FUNCTION; GENETIC RISK; GENETIC SUSCEPTIBILITY; GENOTYPE ENVIRONMENT INTERACTION; HETEROZYGOTE; HIGH RISK POPULATION; HUMAN; HYPERKERATOSIS; LONG TERM EXPOSURE; MALE; PRECANCER; PRIORITY JOURNAL; RISK ASSESSMENT; SKIN CARCINOGENESIS; SKIN CARCINOMA; SKIN DEFECT; STATISTICAL SIGNIFICANCE; XPD GENE;

EID: 0038732377     PISSN: 03784274     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0378-4274(03)00117-6     Document Type: Article

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