Mitochondrial dysfunction leads to telomere attrition and genomic instability.

Aging cell

Volumn 1, Issue 1, 2002, Pages 40-46

  Liu, Lin ^a   Trimarchi, James R ^a   Smith, Peter J S ^a   Keefe, David L ^a  

^a WOMEN AND INFANTS HOSPITAL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIOXIDANT; REACTIVE OXYGEN METABOLITE;

ANIMAL; APOPTOSIS; ARTICLE; C3H MOUSE; C57BL MOUSE; CELL AGING; CELL DIVISION; CELL NUCLEUS; CELL NUCLEUS TRANSPLANTATION; CHROMOSOME BREAKAGE; CYTOLOGY; DRUG EFFECT; FEMALE; GENETICS; MALE; METABOLISM; MITOCHONDRION; MOUSE; OXIDATIVE STRESS; TELOMERE; TISSUE TRANSPLANTATION; ZYGOTE;

ANIMALS; ANTIOXIDANTS; APOPTOSIS; CELL AGING; CELL DIVISION; CELL NUCLEUS; CHROMOSOME BREAKAGE; FEMALE; MALE; MICE; MICE, INBRED C3H; MICE, INBRED C57BL; MITOCHONDRIA; NUCLEAR TRANSFER TECHNIQUES; OXIDATIVE STRESS; REACTIVE OXYGEN SPECIES; TELOMERE; TISSUE TRANSPLANTATION; ZYGOTE;

EID: 0038331337     PISSN: 14749718     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1474-9728.2002.00004.x     Document Type: Article

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