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Volumn 17, Issue 10, 2003, Pages 1337-1339
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Nitric oxide induces polarization of actin in encephalitogenic T cells and inhibits their in vitro trans-endothelial migration in a p70S6 kinase-independent manner.
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Author keywords
[No Author keywords available]
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Indexed keywords
1-HYDROXY-2-OXO-3,3-BIS(2-AMINOETHYL)-1-TRIAZENE;
3,3 BIS(2 AMINOETHYL) 1 HYDROXY 2 OXOTRIAZENE;
ACTIN;
CELL ADHESION MOLECULE;
NITRIC OXIDE;
NITRIC OXIDE DONOR;
S6 KINASE;
TRIAZENE DERIVATIVE;
ALLERGIC ENCEPHALOMYELITIS;
ANIMAL;
ARTICLE;
BIOLOGICAL MODEL;
BRAIN;
CELL CULTURE;
CELL DEATH;
CELL LINE;
CELL MOTION;
CELL POLARITY;
CYTOLOGY;
DOG;
DRUG EFFECT;
ENDOTHELIUM;
IMMUNOLOGY;
LYMPHOCYTE ACTIVATION;
METABOLISM;
MICROFILAMENT;
PHYSIOLOGY;
RAT;
STRESS FIBER;
T LYMPHOCYTE;
TIGHT JUNCTION;
ULTRASTRUCTURE;
ACTINS;
ANIMALS;
BRAIN;
CELL ADHESION MOLECULES;
CELL DEATH;
CELL LINE;
CELL MOVEMENT;
CELL POLARITY;
CELLS, CULTURED;
DOGS;
ENCEPHALOMYELITIS, AUTOIMMUNE, EXPERIMENTAL;
ENDOTHELIUM;
LYMPHOCYTE ACTIVATION;
MICROFILAMENTS;
MODELS, IMMUNOLOGICAL;
NITRIC OXIDE;
NITRIC OXIDE DONORS;
RATS;
RIBOSOMAL PROTEIN S6 KINASES, 70-KDA;
STRESS FIBERS;
T-LYMPHOCYTES;
TIGHT JUNCTIONS;
TRIAZENES;
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EID: 0038265435
PISSN: None
EISSN: 15306860
Source Type: Journal
DOI: 10.1096/fj.02-0577fje Document Type: Article |
Times cited : (24)
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References (0)
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