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New England Journal of Medicine
Volumn 348, Issue 25, 2003, Pages 2583-2584
Interleukin-1-receptor antagonist in the Muckle-Wells syndrome [8]
Author keywords

[No Author keywords available]
Indexed keywords

ANTIHISTAMINIC AGENT; AZATHIOPRINE; CHLORAMBUCIL; COLCHICINE; CORTICOSTEROID; DAPSONE; INFLIXIMAB; INTERLEUKIN 1; INTERLEUKIN 1 RECEPTOR BLOCKING AGENT; MYCOPHENOLIC ACID 2 MORPHOLINOETHYL ESTER; RECOMBINANT INTERLEUKIN 1 RECEPTOR BLOCKING AGENT; SERUM AMYLOID A; THALIDOMIDE;
EID: 0037792866     PISSN: 00284793     EISSN: None     Source Type: Journal    
DOI: 10.1056/NEJM200306193482523     Document Type: Letter
Times cited : (409)
References (5)
  • 1
    • 0035179970 scopus 로고    scopus 로고
    • Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle-Wells syndrome
    • Hoffman HM, Mueller JL, Broide DH, Wanderer AA, Kolodner RD. Mutation ofa new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle-Wells syndrome. Nat Genet 2001;29:301-5.
    • (2001) Nat Genet , vol.29 , pp. 301-305
    • Hoffman, H.M.1    Mueller, J.L.2    Broide, D.H.3    Wanderer, A.A.4    Kolodner, R.D.5
  • 2
    • 0036899758 scopus 로고    scopus 로고
    • De novo CIAS1 mutations, cytokine activation, and evidence for genetic heterogeneity in patients with neonatal-onset multisystem inflammatory disease (NOMID): A new member of the expanding family of pyrinassociated autoinflammatory diseases
    • Aksentijevich I, Nowak M, Mallah M, et al. De novo CIAS1 mutations, cytokine activation, and evidence for genetic heterogeneity in patients with neonatal-onset multisystem inflammatory disease (NOMID): a new member of the expanding family of pyrinassociated autoinflammatory diseases. Arthritis Rheum 2002;46:3340-8.
    • (2002) Arthritis Rheum , vol.46 , pp. 3340-3348
    • Aksentijevich, I.1    Nowak, M.2    Mallah, M.3
  • 3
    • 0036745064 scopus 로고    scopus 로고
    • Association of mutations in the NALP3/CIAS1/PYPAF1 gene with a broad phenotype including recurrent fever, cold sensitivity, sensorineural deafness, and AA amyloidosis
    • Erratum, Arthritis Rheum 2002;46:3398
    • Aganna E, Martinon F, Hawkins PN, et al. Association ofmutations in the NALP3/CIAS1/PYPAF1 gene with a broad phenotype including recurrent fever, cold sensitivity, sensorineural deafness, and AA amyloidosis. Arthritis Rheum 2002;46:2445-52. [Erratum, Arthritis Rheum 2002;46:3398.]
    • (2002) Arthritis Rheum , vol.46 , pp. 2445-2452
    • Aganna, E.1    Martinon, F.2    Hawkins, P.N.3
  • 4
    • 0035822274 scopus 로고    scopus 로고
    • Amyloid load and clinical outcome in AA amyloidosis in relation to circulating concentration of serum amyloid A protein
    • Gillmore JD, Lovat LB, Persey MR, Pepys MB, Hawkins PM. Amyloid load and clinical outcome in AA amyloidosis in relation to circulating concentration of serum amyloid A protein. Lancet 2001;358:24-9.
    • (2001) Lancet , vol.358 , pp. 24-29
    • Gillmore, J.D.1    Lovat, L.B.2    Persey, M.R.3    Pepys, M.B.4    Hawkins, P.M.5
  • 5
    • 0036190776 scopus 로고    scopus 로고
    • Treatment of rheumatoid arthritis with anakinra, a recombinant human interleukin-1 receptor antagonist, in combination with methotrexate: Results of a twentyfour-week, multicenter, randomized, double-blind, placebo-controlled trial
    • Cohen S, Hurd E, Cush J, et al. Treatment of rheumatoid arthritis with anakinra, a recombinant human interleukin-1 receptor antagonist, in combination with methotrexate: results of a twentyfour-week, multicenter, randomized, double-blind, placebo-controlled trial. Arthritis Rheum 2002;46:614-24.
    • (2002) Arthritis Rheum , vol.46 , pp. 614-624
    • Cohen, S.1    Hurd, E.2    Cush, J.3

* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.