Molecular call-and-response: How Salmonella learns the gospel from its host

Trends in Microbiology

Volumn 11, Issue 6, 2003, Pages 245-246

  Dangl, Jeffery L ^a  

^a UNIVERSITY OF NORTH CAROLINA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN; PROTEIN NRAMP; UNCLASSIFIED DRUG;

CYTOLOGY; GENETIC TRANSCRIPTION; GENOTYPE; HOST PATHOGEN INTERACTION; MOLECULAR DYNAMICS; NONHUMAN; PATHOGENICITY; PRIORITY JOURNAL; RECEPTOR UPREGULATION; SALMONELLA ENTERICA; SHORT SURVEY;

NEGIBACTERIA; SALMONELLA; SALMONELLA ENTERICA;

EID: 0037665943     PISSN: 0966842X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0966-842X(03)00127-6     Document Type: Short Survey

References (4)

1. Blackwell J.M., et al. SLC11A1 (formerly NRAMP1) and disease resistance. Cell. Microbiol. 3:2001;773-784.
2. Vidal S.M., et al. Natural resistance to infection with intracellular parasites: isolation of a candidate for Bcg. Cell. 73:1993;469-485.
3. Zaharik M.L., et al. Host-pathogen interactions: host resistance Nramp1 up-regulates the expression of pathogenicity island-2 virulence genes. Proc. Natl. Acad. Sci. U. S. A. 99:2002;15705-15710.
4. Vidal S., et al. The Ity/Lsh/Bcg locus: natural resistance to infection with intracellular parasites is abrogated by disruption of the Nramp1 gene. J. Exp. Med. 182:1995;655-666.