Overexpression of the stress protein Grp94 reduces cardiomyocyte necrosis due to calcium overload and simulated ischemia.

The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Volumn 17, Issue 8, 2003, Pages 923-925

  Vitadello, Maurizio ^a   Penzo, Daniele ^a   Petronilli, Valeria ^a   Michieli, Genny ^a   Gomirato, Selena ^a   Menabò, Roberta ^a   Di Lisa, Fabio ^a   Gorza, Luisa ^a  

^a CNR   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIMYCIN; CALCIUM; GLUCOSE REGULATED PROTEINS; GLUCOSE-REGULATED PROTEINS; HEAT SHOCK PROTEIN 70; IONOPHORE; MEMBRANE PROTEIN;

ANIMAL; ARTICLE; CELL LINE; DRUG EFFECT; GENETIC TRANSFECTION; GENETICS; HEART MUSCLE CELL; HEART MUSCLE ISCHEMIA; HOMEOSTASIS; METABOLISM; MOUSE; NECROSIS; PATHOLOGY; RAT; WESTERN BLOTTING;

ANIMALS; BLOTTING, WESTERN; CALCIMYCIN; CALCIUM; CELL LINE; HOMEOSTASIS; HSP70 HEAT-SHOCK PROTEINS; IONOPHORES; MEMBRANE PROTEINS; MICE; MYOCARDIAL ISCHEMIA; MYOCYTES, CARDIAC; NECROSIS; RATS; TRANSFECTION;

MLCS; MLOWN;

EID: 0037646962     PISSN: None     EISSN: 15306860     Source Type: Journal    
DOI: 10.1096/fj.02-0644fje     Document Type: Article

References (0)