AMPK-β1 subunit is a p53-independent stress responsive protein that inhibits tumor cell growth upon forced expression

Carcinogenesis

Volumn 24, Issue 5, 2003, Pages 827-834

  Li, Jun ^a,b   Jiang, Ping ^a   Robinson, Megan ^a   Lawrence, Theodore S ^b   Sun, Yi ^a  

^a PFIZER INC   (United States)

^b UNIVERSITY OF MICHIGAN   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYCLIC AMP DEPENDENT PROTEIN KINASE; ETOPOSIDE; PROTEIN P53; AMP ACTIVATED PROTEIN KINASE BETA; AMP-ACTIVATED PROTEIN KINASE BETA; PROTEIN KINASE; PROTEIN SUBUNIT;

APOPTOSIS; BINDING SITE; CANCER CELL; CANCER INHIBITION; CELL GROWTH; CELL KILLING; CHROMATIN; CONTROLLED STUDY; DNA DAMAGE; ENZYME SUBUNIT; GENE EXPRESSION; GENE IDENTIFICATION; GENE INDUCTION; GENE OVEREXPRESSION; GENE REPRESSION; GENE TARGETING; GROWTH INHIBITION; HUMAN; HUMAN CELL; HUMAN TISSUE; IMMUNOPRECIPITATION; IN VITRO STUDY; IN VIVO STUDY; INTRON; LUNG CARCINOMA; NORTHERN BLOTTING; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN EXPRESSION; REVIEW; SIGNAL TRANSDUCTION; STRESS; TEMPERATURE; ARTICLE; BIOSYNTHESIS; CELL CULTURE; COLD; DNA MICROARRAY; DRUG EFFECT; GEL MOBILITY SHIFT ASSAY; GENE EXPRESSION REGULATION; GENETIC TRANSFECTION; GENETICS; LUNG TUMOR; METABOLISM; MOLECULAR GENETICS; PATHOLOGY; POLYMERASE CHAIN REACTION; TRANSACTIVATION; UPREGULATION; WESTERN BLOTTING;

APOPTOSIS; BASE SEQUENCE; BLOTTING, WESTERN; COLD; ELECTROPHORETIC MOBILITY SHIFT ASSAY; ETOPOSIDE; GENE EXPRESSION REGULATION, NEOPLASTIC; HUMANS; LUNG NEOPLASMS; MOLECULAR SEQUENCE DATA; OLIGONUCLEOTIDE ARRAY SEQUENCE ANALYSIS; POLYMERASE CHAIN REACTION; PROMOTER REGIONS (GENETICS); PROTEIN KINASES; PROTEIN SUBUNITS; TRANS-ACTIVATION (GENETICS); TRANSFECTION; TUMOR CELLS, CULTURED; TUMOR SUPPRESSOR PROTEIN P53; UP-REGULATION;

EID: 0037634018     PISSN: 01433334     EISSN: None     Source Type: Journal    
DOI: 10.1093/carcin/bgg032     Document Type: Review

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