Familial hyperactivity of angiotensin-converting enzyme (ACE);Familiaire hyperactiviteit van angiotensineconverterend enzym (ACE)

Nederlands Tijdschrift voor Geneeskunde

Volumn 147, Issue 11, 2003, Pages 507-510

  Kramers, C ^a   Adema, G J ^a   Korte, M ^b   Deinum, J ^a  

^a RADBOUD UNIVERSITY MEDICAL CENTER   (Netherlands)

^b Medisch Centrum Rijnmond Zuid   (Netherlands)

Author keywords

[No Author keywords available]

Indexed keywords

ALKALINE PHOSPHATASE; DIPEPTIDYL CARBOXYPEPTIDASE;

ADULT; ARTHRALGIA; ARTICLE; CLINICAL ARTICLE; DYSPNEA; ENZYME ACTIVITY; ENZYME BLOOD LEVEL; FAMILIAL DISEASE; FATIGUE; FEMALE; HUMAN; MALE; NEPHROLITHIASIS; NEUROLOGIC DISEASE; POINT MUTATION; SARCOIDOSIS;

ADULT; AGED; DIAGNOSIS, DIFFERENTIAL; FEMALE; HUMANS; MALE; METABOLISM, INBORN ERRORS; MIDDLE AGED; PEDIGREE; PEPTIDYL-DIPEPTIDASE A; POINT MUTATION; SARCOIDOSIS;

EID: 0037443945     PISSN: 00282162     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (10)

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9. Woodman ZL, Oppong SY, Cook S, Hooper NM, Schwager SL, Brandt WF, et al. Shedding of somatic angiotensin-converting enzyme (ACE) is inefficient compared with testis ACE despite cleavage at identical stalk sites. Biochem J 2000;347 Pt 3:711-8.
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