Senescence, aging, and malignant transformation mediated by p53 in mice lacking the brca1 full-length isoform

Genes and Development

Volumn 17, Issue 2, 2003, Pages 201-213

  Cao, Liu ^a   Li, Wenmei ^a   Kim, Sangsoo ^a   Brodie, Steven G ^a   Deng, Chu Xia ^a  

^a NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES   (United States)

Author keywords

Aging; p21; p53; Senescence; Tumorigenesis

Indexed keywords

BRCA1 PROTEIN; DNA; PROTEIN P21; PROTEIN P53;

AGED; AGING; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BREAST TUMOR; CELL AGING; CELL CYCLE; CONTROLLED STUDY; DNA DAMAGE; GENE OVEREXPRESSION; LIFESPAN; MALIGNANT TRANSFORMATION; MOUSE; NONHUMAN; OSTEOPOROSIS; PHENOTYPE; PRIORITY JOURNAL; SENESCENCE; SKIN ATROPHY; TUMOR SUPPRESSOR GENE; WOUND HEALING;

AGING; AGING, PREMATURE; ANIMALS; BRCA1 PROTEIN; CELL AGING; FEMALE; GENES, BRCA1; GENES, P53; HAPLOIDY; MALE; MICE; MICE, MUTANT STRAINS; NEOPLASMS, EXPERIMENTAL; PHENOTYPE; PROTEIN ISOFORMS;

ANIMALIA;

EID: 0037439071     PISSN: 08909369     EISSN: None     Source Type: Journal    
DOI: 10.1101/gad.1050003     Document Type: Article

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