TERT suppresses apoptotis at a premitochondrial step by a mechanism requiring reverse transcriptase activity and 14-3-3 protein-binding ability.

The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Volumn 17, Issue 6, 2003, Pages 767-769

  Zhang, Peisu ^a   Chan, Sic L ^a   Fu, Weiming ^a   Mendoza, Marty ^a   Mattson, Mark P ^a  

^a NATIONAL INSTITUTE ON AGING   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANTINEOPLASTIC AGENT; APOPTOSIS INDUCING FACTOR; CYTOCHROME C; DNA BINDING PROTEIN; ENZYME INHIBITOR; ETOPOSIDE; FLAVOPROTEIN; MEMBRANE PROTEIN; PDCD8 PROTEIN, HUMAN; PROTEIN 14 3 3; RNA DIRECTED DNA POLYMERASE; STAUROSPORINE; TELOMERASE; TERT PROTEIN, HUMAN; TYROSINE 3 MONOOXYGENASE;

APOPTOSIS; ARTICLE; BINDING SITE; CELL LINE; CELL NUCLEUS; DRUG EFFECT; GENETICS; HELA CELL; HUMAN; METABOLISM; MITOCHONDRION; MUTATION; PHYSIOLOGY; PROTEIN BINDING; TRANSPORT AT THE CELLULAR LEVEL;

14-3-3 PROTEINS; ANTINEOPLASTIC AGENTS, PHYTOGENIC; APOPTOSIS; APOPTOSIS INDUCING FACTOR; BINDING SITES; BIOLOGICAL TRANSPORT; CELL LINE; CELL NUCLEUS; CYTOCHROME C GROUP; DNA-BINDING PROTEINS; ENZYME INHIBITORS; ETOPOSIDE; FLAVOPROTEINS; HELA CELLS; HUMANS; MEMBRANE PROTEINS; MITOCHONDRIA; MUTATION; PROTEIN BINDING; RNA-DIRECTED DNA POLYMERASE; STAUROSPORINE; TELOMERASE; TYROSINE 3-MONOOXYGENASE;

MLCS; MLOWN;

EID: 0037390962     PISSN: None     EISSN: 15306860     Source Type: Journal    
DOI: 10.1096/fj.02-0603fje     Document Type: Article

References (0)