Coronary flow-induced inotropism is modulated by binding of dextrans to the endothelial luminal surface

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 284, Issue 4 53-4, 2003, Pages

  Gonzalez Castillo, Carmen ^a   Rubio, Rafael ^a   Zenteno Savin, Tania ^b  

^a UNIVERSIDAD AUTÓNOMA DE SAN LUIS POTOSÍ   (Mexico)

^b CENTRO DE INVESTIGACIONES BIOLÓGICAS DEL NOROESTE   (Mexico)

Author keywords

Albumin; Dextranase; Endothelial glycocalyx; Endothelial lectins; Interstitial volume; Irreversibility; Protein polymerization; Shearing stress; Steady state

Indexed keywords

DEXTRAN DERIVATIVE; DEXTRANASE;

ANIMAL TISSUE; ARTICLE; BLOOD VESSEL WALL; CONCENTRATION RESPONSE; CONTROLLED STUDY; CORONARY ARTERY BLOOD FLOW; DRUG BINDING SITE; DRUG HYDROLYSIS; GUINEA PIG; INOTROPISM; MALE; MOLECULAR WEIGHT; NONHUMAN; PRIORITY JOURNAL; VASCULAR ENDOTHELIUM; VISCOSITY;

EID: 0037378341     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.00323.2002     Document Type: Article

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