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Volumn 14, Issue 3, 2003, Pages 320-322

Is restoration of intracellular trafficking clinically feasible in the long QT syndrome?: The example of HERG mutations

Author keywords

[No Author keywords available]

Indexed keywords

1 [2 (6 METHYL 2 PYRIDYL)ETHYL] 4 (4 METHYLSULFONYLAMINOBENZOYL)PIPERIDINE; 4 PHENYLBUTYRIC ACID; ASTEMIZOLE; CISAPRIDE; POTASSIUM CHANNEL; THAPSIGARGIN; TRANSMEMBRANE CONDUCTANCE REGULATOR;

EID: 0037359570     PISSN: 10453873     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1540-8167.2003.02363.x     Document Type: Review
Times cited : (13)

References (18)
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    • Increased risk of arrhythmic events in long-QT syndrome with mutations in the pore region of the human ether-a-go-go-related gene potassium channel
    • Moss AJ, Zareba W, Kaufman ES, et al: Increased risk of arrhythmic events in long-QT syndrome with mutations in the pore region of the human ether-a-go-go-related gene potassium channel. Circulation 2002;105:794-799.
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    • Long-QT syndrome-associated mutations in the Per-Arnt-Sim (PAS) domain of HERG potassium channels accelerate channel deactivation
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  • 7
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  • 8
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.