Bioassay of endothelium-derived hyperpolarizing factor with abolishment of nitric oxide and the role of gap junctions in the porcine coronary circulation

Drug Development Research

Volumn 58, Issue 1, 2003, Pages 99-110

  Yang, Qin ^a   Ge, Zhi Dong ^b   Yang, Cheng Qin ^b   Huang, Yu ^a   He, Guo Wei ^a,b,c  

^a CHINESE UNIVERSITY OF HONG KONG   (Hong Kong)

^b OREGON HEALTH AND SCIENCE UNIVERSITY   (United States)

^c CHINESE UNIVERSITY OF HONG KONG   (Hong Kong)

Author keywords

Circulatory physiology; Coronary circulation; EDHF; Endothelial factors; Gap junctions; K channel; Nitric oxide

Indexed keywords

18ALPHA GLYCYRRHETINIC ACID; APAMIN; BRADYKININ; CHARYBDOTOXIN; ENDOTHELIUM DERIVED HYPERPOLARIZING FACTOR; GLIBENCLAMIDE; HEMOGLOBIN; HEPTANOL; INDOMETACIN; N(G) NITROARGININE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE INHIBITOR; POTASSIUM CHANNEL; PROSTAGLANDIN SYNTHASE INHIBITOR; SCAVENGER;

ANIMAL EXPERIMENT; ANIMAL TISSUE; BIOASSAY; BLOOD VESSEL REACTIVITY; CONFERENCE PAPER; CONTROLLED STUDY; CORONARY ARTERY; DOSE RESPONSE; ELECTROCHEMICAL ANALYSIS; ENDOTHELIUM; GAP JUNCTION; HYPERPOLARIZATION; MICROCIRCULATION; MICROELECTRODE; MYOGRAPHY; NONHUMAN; SWINE; VASODILATATION;

EID: 0037282441     PISSN: 02724391     EISSN: None     Source Type: Journal    
DOI: 10.1002/ddr.10137     Document Type: Conference Paper

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