Possible involvement of phospholipase D and protein kinase C in elevated glucose concentration induced vascular growth
Yasunari K, Kohno M, Kano H, Yokokawa K, Horio T, Yoshikawa J. Possible involvement of phospholipase D and protein kinase C in elevated glucose concentration induced vascular growth. Hypertension 1996;28:159-68.
Mechanisms of action of troglitazone in the prevention of high glucose-induced migration and proliferation of cultured coronary smooth muscle cells
Yasunari K, Kohno M, Kano H, Yokokawa K, Minami M, Yoshikawa J. Mechanisms of action of troglitazone in the prevention of high glucose-induced migration and proliferation of cultured coronary smooth muscle cells. Circ Res 1997;81:953-62.
Antioxidants improve impaired insulin-mediated glucose uptake and prevent migration and proliferation of cultured rabbit coronary smooth muscle cells induced by high glucose
Yasunari K, Kohno M, Kano H, Minami M, Yokokawa K, Yoshikawa J. Antioxidants improve impaired insulin-mediated glucose uptake and prevent migration and proliferation of cultured rabbit coronary smooth muscle cells induced by high glucose. Circulation 1999;99:1370-8.
Plasma adrenaline, noradrenaline and dopamine in man and different animal species
Bühler HU, Prada MD, Haefely W, Picotti GB. Plasma adrenaline, noradrenaline and dopamine in man and different animal species. J Physiol 1978;276:311-20.
Free and conjugated plasma catecholamines in hypertensive patients with or without pheochromocytoma
Kuchel O, Buu NT, Fontaine A, et al. Free and conjugated plasma catecholamines in hypertensive patients with or without pheochromocytoma. Hypertension 1980;2:177-86.
Is there a third peripheral catecholaminergic system? Endogenous dopamine as an autocrine/paracrine substance derived plasma DOPA and inactivated by conjugation
Goldstein DS, Mezey E, Yamamoto T, Aneman A, Friberg P, Eisenhofer G. Is there a third peripheral catecholaminergic system? Endogenous dopamine as an autocrine/paracrine substance derived plasma DOPA and inactivated by conjugation. Hypertens Res 1995; 18(suppl I):S93-9.
Angiotensin-converting enzyme inhibitor versus angiotensin II, AT1 receptor antagonist: Effects on smooth muscle cell migration and proliferation after balloon catheter injury
Prescott MF, Webb RL, Reidy MA. Angiotensin-converting enzyme inhibitor versus angiotensin II, AT1 receptor antagonist: Effects on smooth muscle cell migration and proliferation after balloon catheter injury. Am J Pathol 1991;139:1291-96.