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Volumn 444, Issue 1-2, 2002, Pages 115-121

Nicotine suppresses gastric wound repair via the inhibition of polyamine and K+ channel expression

Author keywords

Cell migration; Cell proliferation; K+ channel; Nicotine; Nicotine receptor; Ornithine decarboxylase; Wound healing

Indexed keywords

MECAMYLAMINE; NICOTINE; ORNITHINE DECARBOXYLASE; POTASSIUM CHANNEL; SPERMIDINE; POLYAMINE;

EID: 0037165831     PISSN: 00142999     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0014-2999(02)01610-2     Document Type: Article
Times cited : (27)

References (47)
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    • Konno, S.1    Chiao, J.W.2    Wu, J.M.3
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    • Ionic events induced by epidermal growth factor. Evidence that hyperpolarization and stimulated cation influx play a role in the stimulation of cell growth
    • (1991) J. Biol. Chem. , vol.266 , pp. 6329-6335
    • Magni, M.1    Meldolesi, J.2    Pandiella, A.3
  • 41
    • 0026113581 scopus 로고
    • A 23-pS Ca2(+)-activated K+ channel in MCF-7 human breast carcinoma cells: An apparent correlation of channel incidence with the rate of cell proliferation
    • (1991) Pfluegers Arch. , vol.417 , pp. 562-570
    • Wegman, E.A.1    Young, J.A.2    Cook, D.I.3


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.