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Volumn 444, Issue 1-2, 2002, Pages 115-121
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Nicotine suppresses gastric wound repair via the inhibition of polyamine and K+ channel expression
a a a a a a |
Author keywords
Cell migration; Cell proliferation; K+ channel; Nicotine; Nicotine receptor; Ornithine decarboxylase; Wound healing
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Indexed keywords
MECAMYLAMINE;
NICOTINE;
ORNITHINE DECARBOXYLASE;
POTASSIUM CHANNEL;
SPERMIDINE;
POLYAMINE;
ACUTE TOXICITY;
ANIMAL CELL;
ARTICLE;
CELL MIGRATION;
CELL PROLIFERATION;
CONTROLLED STUDY;
DRUG ANTAGONISM;
ENZYME INACTIVATION;
EPITHELIUM CELL;
MIGRATION INHIBITION;
MITOSIS INHIBITION;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
RAT;
WOUND HEALING;
WOUND HEALING IMPAIRMENT;
ANIMAL;
CELL MOTION;
DRUG EFFECT;
PHYSIOLOGY;
STOMACH MUCOSA;
ANIMAL;
CELL MOVEMENT;
GASTRIC MUCOSA;
NICOTINE;
ORNITHINE DECARBOXYLASE;
POLYAMINES;
POTASSIUM CHANNELS;
RATS;
SUPPORT, NON-U.S. GOV'T;
WOUND HEALING;
ANIMALS;
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EID: 0037165831
PISSN: 00142999
EISSN: None
Source Type: Journal
DOI: 10.1016/S0014-2999(02)01610-2 Document Type: Article |
Times cited : (27)
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References (47)
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