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Volumn 277, Issue 29, 2002, Pages 26699-26707
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A novel p53 transcriptional repressor element (p53TRE) and the asymmetrical contribution of two p53 binding sites modulate the response of the placental transforming growth factor-β promoter to p53
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Author keywords
[No Author keywords available]
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Indexed keywords
BIOASSAY;
DNA;
ELECTROPHORESIS;
GROWTH KINETICS;
RNA;
SIGNALING;
TRANSCIPTIONAL REPRESSOR ELEMENT;
BIOCHEMISTRY;
MESSENGER RNA;
P53 TRANSCRIPTIONAL REPRESSOR ELEMENT;
PLACENTAL TRANSFORMING GROWTH FACTOR BETA;
PROTEIN P21;
PROTEIN P53;
REPRESSOR PROTEIN;
TRANSFORMING GROWTH FACTOR BETA;
UNCLASSIFIED DRUG;
ARTICLE;
BINDING SITE;
CONTROLLED STUDY;
DOWN REGULATION;
ELECTROPHORETIC MOBILITY;
HUMAN;
HUMAN CELL;
KINETICS;
MUTATIONAL ANALYSIS;
PRIORITY JOURNAL;
PROMOTER REGION;
PROTEIN BINDING;
PROTEIN EXPRESSION;
PROTEIN FUNCTION;
TRANSACTIVATION;
TRANSCRIPTION REGULATION;
TUMOR CELL LINE;
BASE SEQUENCE;
BINDING SITES;
CYCLIN-DEPENDENT KINASE INHIBITOR P21;
CYCLINS;
DNA MUTATIONAL ANALYSIS;
DOWN-REGULATION;
FEMALE;
GROWTH SUBSTANCES;
HELA CELLS;
HUMANS;
MOLECULAR SEQUENCE DATA;
PREGNANCY PROTEINS;
PROMOTER REGIONS (GENETICS);
REPRESSOR PROTEINS;
RNA, MESSENGER;
STRUCTURE-ACTIVITY RELATIONSHIP;
TRANS-ACTIVATION (GENETICS);
TUMOR CELLS, CULTURED;
TUMOR SUPPRESSOR PROTEIN P53;
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EID: 0037135581
PISSN: 00219258
EISSN: None
Source Type: Journal
DOI: 10.1074/jbc.M203020200 Document Type: Article |
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Times cited : (22)
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References (54)
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