Effects on splicing and protein function of three mutations in codon N296 of tau in vitro

Neuroscience Letters

Volumn 323, Issue 1, 2002, Pages 33-36

  Grover, Andrew ^a,b   Deurel, Michael ^a   Yen, Shu Hui ^a   Hutton, Mike ^a  

^a MAYO CLINIC   (United States)

^b UNIVERSITY OF BIRMINGHAM   (United Kingdom)

Author keywords

Aggregation; Fronto temporal dementia; Microtubule; Mutation; Splicing; Tau

Indexed keywords

TAU PROTEIN;

ARTICLE; CHROMOSOME 17; CODON; EXON; FRONTOTEMPORAL DEMENTIA; GENE MUTATION; HETEROZYGOSITY; HOMOZYGOSITY; IDIOPATHIC DISEASE; IN VITRO STUDY; MICROTUBULE ASSEMBLY; PARKINSON DISEASE; PARKINSONISM; PATHOGENICITY; PHENOTYPE; PRIORITY JOURNAL; PROGRESSIVE SUPRANUCLEAR PALSY; PROTEIN AGGREGATION; PROTEIN FUNCTION; RNA SPLICING;

EID: 0037134098     PISSN: 03043940     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0304-3940(02)00124-6     Document Type: Article

References (14)

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14. Self-assembly of the brain MAP-2 microtubule-binding region into polymeric structures resembling Alzheimer filaments