The stress-activated protein kinases p38 alpha and JNK1 stabilize p21(Cip1) by phosphorylation.

The Journal of biological chemistry

Volumn 277, Issue 33, 2002, Pages 29792-29802

  Kim, Geum Yi ^a   Mercer, Stephen E ^a   Ewton, Daina Z ^a   Yan, Zhongfa ^a   Jin, Kideok ^a   Friedman, Eileen ^a  

^a State University of New York Upstate Medical University: College of Medicine   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

4 (4 FLUOROPHENYL) 2 (4 METHYLSULFINYLPHENYL) 5 (4 PYRIDYL)IMIDAZOLE; CYCLIN DEPENDENT KINASE INHIBITOR 1A; CYCLINE; IMIDAZOLE DERIVATIVE; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE 14; PYRIDINE DERIVATIVE; SERINE; STRESS ACTIVATED PROTEIN KINASE 1; THREONINE; TRANSFORMING GROWTH FACTOR BETA;

ARTICLE; CELL CULTURE; METABOLISM; PHOSPHORYLATION; SIGNAL TRANSDUCTION; UPREGULATION;

CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLINS; IMIDAZOLES; MITOGEN-ACTIVATED PROTEIN KINASE 14; MITOGEN-ACTIVATED PROTEIN KINASE 8; MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORYLATION; PYRIDINES; SERINE; SIGNAL TRANSDUCTION; THREONINE; TRANSFORMING GROWTH FACTOR BETA; TUMOR CELLS, CULTURED; UP-REGULATION;

EID: 0037119470     PISSN: 00219258     EISSN: None     Source Type: Journal    
DOI: 10.1074/jbc.M201299200     Document Type: Article

References (0)