Open mind, open airways: Broadening the paradigm of prostaglandins and allergic airway inflammation

American Journal of Respiratory and Critical Care Medicine

Volumn 165, Issue 8, 2002, Pages 1035-1036

  Peters Golden, Marc ^a  

^a UNIVERSITY OF MICHIGAN HEALTH SYSTEM   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYCLOOXYGENASE 1; CYCLOOXYGENASE 2; CYTOKINE; INTERLEUKIN 13; INTERLEUKIN 4; INTERLEUKIN 5; PROSTAGLANDIN;

ALLERGIC ASTHMA; ALLERGY; BRONCHUS HYPERREACTIVITY; CYTOKINE PRODUCTION; EDITORIAL; ENZYME INDUCTION; HUMAN; IMMUNE RESPONSE; PRIORITY JOURNAL; RESPIRATORY TRACT INFLAMMATION; TH2 CELL;

EID: 0037090713     PISSN: 1073449X     EISSN: None     Source Type: Journal    
DOI: 10.1164/ajrccm.165.8.2202071c     Document Type: Editorial

References (10)

1. Selective cyclooxygenase-1 and -2 inhibitors each increase allergic inflammation and airway hyperresponsiveness in mice
2. 2 as a mediator of allergic asthma
3. Peroxisome proliferator-activated receptors α and γ are activated by indomethacin and other non-steroidal anti-inflammatory drugs
4. Mixed messages: Modulation of inflammation and immune responses by prostglandins and thromboxanes
5. 2 inhibits production of Th1 lymphokines but not of Th2 lymphokines
6. cAMP-elevating agents suppress dendritic cell function
7. 2
8. Cyclooxygenase-2 expression during allergic inflammation in guinea-pig lungs
9. 2 and to express cyclooxygenase-2
10. Inhibition of cyclooxygenase-2 aggravates doxorubicin-mediated cardiac injury in vivo