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Volumn 32, Issue 4, 2002, Pages 370-374
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Hypothesis: The mitochondrial NO· signaling pathway, and the transduction of nitrosative to oxidative cell signals: An alternative function for cytochrome c oxidase
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Author keywords
Cell signaling; Free radicals; Hydrogen peroxide; Mitochondria; Nitric oxide
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Indexed keywords
ADENOSINE TRIPHOSPHATE;
AMINO ACID;
CASPASE;
CYSTEINE;
CYTOCHROME C OXIDASE;
FREE RADICAL;
GUANYLATE CYCLASE;
HEMOPROTEIN;
HYDROGEN PEROXIDE;
METALLOPROTEIN;
NITRIC OXIDE;
OXYGEN;
APOPTOSIS;
ARTICLE;
BIOSYNTHESIS;
CELL ACTIVATION;
CELL ACTIVITY;
ENZYME ACTIVITY;
ENZYME BINDING;
MAMMAL CELL;
MITOCHONDRIAL RESPIRATION;
MITOCHONDRION;
MOLECULAR INTERACTION;
NITROSATION;
OXIDATION;
OXIDATION REDUCTION REACTION;
OXIDATIVE STRESS;
PRIORITY JOURNAL;
PROTEIN FUNCTION;
REGULATORY MECHANISM;
SIGNAL TRANSDUCTION;
BIOLOGICAL MODEL;
CELL MEMBRANE POTENTIAL;
DOSE RESPONSE;
METABOLISM;
ADENOSINE TRIPHOSPHATE;
DOSE-RESPONSE RELATIONSHIP, DRUG;
ELECTRON TRANSPORT COMPLEX IV;
FREE RADICALS;
HYDROGEN PEROXIDE;
MEMBRANE POTENTIALS;
MITOCHONDRIA;
MODELS, BIOLOGICAL;
NITRIC OXIDE;
OXIDATION-REDUCTION;
OXYGEN;
SIGNAL TRANSDUCTION;
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EID: 0037082357
PISSN: 08915849
EISSN: None
Source Type: Journal
DOI: 10.1016/S0891-5849(01)00805-X Document Type: Article |
Times cited : (123)
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References (41)
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