Curing the septic diaphragm with the ventilator

American Journal of Respiratory and Critical Care Medicine

Volumn 165, Issue 2, 2002, Pages 145-146

  Laghi, Franco ^a  

^a LOYOLA UNIVERSITY MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

NITRIC OXIDE;

ARTIFICIAL VENTILATION; BREATHING MUSCLE; DIAPHRAGM MUSCLE; EDITORIAL; HUMAN; MUSCLE INJURY; PATHOPHYSIOLOGY; PHRENIC NERVE; PRIORITY JOURNAL; SEPSIS;

EID: 0037080704     PISSN: 1073449X     EISSN: None     Source Type: Journal    
DOI: 10.1164/ajrccm.165.2.2111077a     Document Type: Editorial

References (12)

1. Diaphragm sarcolemmal injury is induced by sepsis and alleviated by nitric oxide synthase inhibition
2. Inducible NO synthase II and neuronal NO synthase I are constitutively expressed in different structures of guinea pig skeletal muscle: Implications for contractile function
3. Lipopolysaccharide-induced diaphragmatic contractile dysfunction and sarcolemmal injury in mice lacking the neuronal nitric oxide synthase
4. Mechanical ventilation protects against diaphragm injury in sepsis. Interaction of oxidative and mechanical stresses
5. Expression of the inducible nitric oxide synthase gene in diaphragm and skeletal muscle
6. Muscular contractile failure in septic patients: Role of the inducible nitric oxide synthase pathway
7. Noninvasive ventilation in immunosuppressed patients with pulmonary infiltrates, fever, and acute respiratory failure
8. Noninvasive ventilation
9. Lipopolysaccharide-induced diaphragmatic contractile dysfunction in mice lacking the inducible nitric oxide synthase
10. Effect of free radical scavengers on endotoxin-induced respiratory muscle dysfunction
11. Sepsis is associated with increased mRNAs of the ubiquitin-proteasome proteolytic pathway in human skeletal muscle
12. Cecal ligation and puncture peritonitis model shows decreased nicotinic acetylcholine receptor numbers in rat muscle: Immunopathologic mechanisms?