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Oncogene

Volumn 21, Issue 2, 2002, Pages 291-298

Suppression of Neu-induced mammary tumor growth in cyclin D1 deficient mice is compensated for by cyclin E

(4) Bowe, Damon B a Kenney, Nicholas J c Adereth, Yair a Maroulakou, Ioanna G a,b

a MEDICAL UNIVERSITY OF SOUTH CAROLINA (United States)

b MEDICAL UNIVERSITY OF SOUTH CAROLINA (United States)

c HAMPTON UNIVERSITY (United States)

Author keywords

Cyclin D1; Cyclin E; Mammary gland; Mice; Neu

Indexed keywords

ANTINEOPLASTIC AGENT; CELL PROTEIN; CYCLIN D1; CYCLIN E; EPIDERMAL GROWTH FACTOR RECEPTOR 2; GENE PRODUCT; TYROSINE KINASE RECEPTOR; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BREAST CARCINOMA; CANCER GROWTH; CARCINOGENESIS; CELL CYCLE; CELL GROWTH; CONTROLLED STUDY; DISEASE ASSOCIATION; FEMALE; GENE AMPLIFICATION; GENE OVEREXPRESSION; GROWTH INHIBITION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION;

ADENOCARCINOMA; ANIMALS; CYCLIN D1; CYCLIN E; FEMALE; GENES, ERBB-2; KINETICS; MAMMARY NEOPLASMS, EXPERIMENTAL; MICE; MICE, KNOCKOUT; RECEPTOR, ERBB-2; SIGNAL TRANSDUCTION; SUPPRESSION, GENETIC;

ANIMALIA;

EID: 0037050256 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205025 Document Type: Article

Times cited : (54)

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