α-thrombin activates Akt via a nonreceptor tyrosine kinase in IIC9 cells

Annals of the New York Academy of Sciences

Volumn 973, Issue , 2002, Pages 142-144

  Phillips Mason, Polly J ^a   Goel, Reema ^a   Baldassare, Joseph J ^a  

^a SAINT LOUIS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Akt pathway; Nonreceptor tyrosice kinase; thrombin

Indexed keywords

EPIDERMAL GROWTH FACTOR; GENISTEIN; PLATELET DERIVED GROWTH FACTOR RECEPTOR; PROTEIN KINASE B; PROTEIN TYROSINE KINASE; PROTEIN TYROSINE KINASE INHIBITOR; RECEPTOR PROTEIN; THROMBIN; TYROSINE KINASE RECEPTOR;

CONFERENCE PAPER; ENZYME ACTIVATION; NONHUMAN; PROTEIN PHOSPHORYLATION; TRANSACTIVATION;

EID: 0036961974     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1111/j.1749-6632.2002.tb04623.x     Document Type: Conference Paper

References (4)

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3. PHILLIPS-MASON, P.J., D.M. RABEN & J.J. BALDASSARE. 2000. Phosphatidylinositol 3-kinase activity regulates alpha-thrombin-stimulated G(1) progression by its effect on cyclin D1 expression and cyclin-dependent kinase 4 activity. J. Biol. Chem. 275: 18046-18053.
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