Overexpression of SOD1 protects vulnerable motor neurons after spinal cord injury by attenuating mitochondrial cytochrome c release.

The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Volumn 16, Issue 14, 2002, Pages 1997-1999

  Sugawara, Taku ^a   Lewén, Anders ^a   Gasche, Yvan ^a   Yu, Fengshan ^a   Chan, Pak H ^a  

^a STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

COPPER ZINC SUPEROXIDE DISMUTASE; CYTOCHROME C; DNA FRAGMENT; SUPEROXIDE; SUPEROXIDE DISMUTASE;

ANIMAL; APOPTOSIS; ARTICLE; BIOLOGICAL MODEL; CELL NUCLEUS; CELL PROTECTION; CHEMISTRY; GENETICS; METABOLISM; MITOCHONDRION; MOTONEURON; PATHOLOGY; PHYSIOLOGY; RAT; SPINAL CORD; SPINAL CORD DISEASE; TRANSGENIC ANIMAL; ULTRASTRUCTURE;

ANIMALS; ANIMALS, GENETICALLY MODIFIED; APOPTOSIS; CELL NUCLEUS; CYTOCHROME C GROUP; CYTOPROTECTION; DNA FRAGMENTATION; MITOCHONDRIA; MODELS, BIOLOGICAL; MOTOR NEURONS; RATS; SPINAL CORD; SPINAL CORD DISEASES; SUPEROXIDE DISMUTASE; SUPEROXIDES;

MLCS; MLOWN;

EID: 0036883891     PISSN: None     EISSN: 15306860     Source Type: Journal    
DOI: 10.1096/fj.02-0251fje     Document Type: Article

References (0)