v-Crk activates the phosphoinositide 3-kinase/AKT pathway by utilizing focal adhesion kinase and H-Ras

Molecular and Cellular Biology

Volumn 22, Issue 20, 2002, Pages 7015-7023

  Akagi, Tsuyoshi ^a   Murata, Kazutaka ^a   Shishido, Tomoyuki ^a   Hanafusa, Hidesaburo ^a  

^a OSAKA BIOSCIENCE INSTITUTE   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

FOCAL ADHESION KINASE; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN KINASE B; PROTEIN P85;

ANIMAL CELL; ARTICLE; CELL TRANSFORMATION; CONTROLLED STUDY; EMBRYO; ENZYME ACTIVATION; FIBROBLAST; GENE FUNCTION; GENE OVEREXPRESSION; MOUSE; NONHUMAN; ONCOGENE; ONCOGENE H RAS; ONCOGENE V CRK; PRIORITY JOURNAL; PROTEIN DOMAIN; REGULATORY MECHANISM; SIGNAL TRANSDUCTION;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ANIMALS; AVIAN SARCOMA VIRUSES; CHICK EMBRYO; ENZYME ACTIVATION; FOCAL ADHESION KINASE 1; FOCAL ADHESION PROTEIN-TYROSINE KINASES; HUMANS; ONCOGENE PROTEIN V-CRK; PHOSPHORYLATION; PROTEIN-SERINE-THREONINE KINASES; PROTEIN-TYROSINE KINASES; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-AKT; PROTO-ONCOGENE PROTEINS P21(RAS); RECOMBINANT FUSION PROTEINS; RETROVIRIDAE PROTEINS, ONCOGENIC; SIGNAL TRANSDUCTION; SRC-FAMILY KINASES; TRANSFORMATION, GENETIC; TYROSINE;

ANIMALIA; AVES; AVIAN SARCOMA VIRUS; GALLUS GALLUS;

EID: 0036787909     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/MCB.22.20.7015-7023.2002     Document Type: Article

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