Increased serum concentrations of tissue plasminogen activator correlate with an adverse clinical outcome in patients with bacterial meningitis [1]

Journal of Neurology Neurosurgery and Psychiatry

Volumn 73, Issue 4, 2002, Pages 456-

  Winkler, F ^a   Kastenbauer, S ^a   Koedel, U ^a   Pfister, H W ^a  

^a LUDWIG MAXIMILIANS UNIVERSITY   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

FIBRIN; PLASMIN; TISSUE PLASMINOGEN ACTIVATOR;

ADULT; AGED; BACTERIAL MENINGITIS; BLOOD BRAIN BARRIER; CEREBROSPINAL FLUID LEVEL; CLINICAL ARTICLE; CONTROLLED STUDY; GENE EXPRESSION; GUILLAIN BARRE SYNDROME; HUMAN; LETTER; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL;

EID: 0036785101     PISSN: 00223050     EISSN: None     Source Type: Journal    
DOI: 10.1136/jnnp.73.4.456     Document Type: Letter

References (5)

1. Pfister HW, Fontana A, Tauber MG, et al. Mechanisms of brain injury in bacterial meningitis: workshop summary. Clin Infect Dis 1994;19:463-79.
2. Strickland S. Tissue plasminogen activator in nervous system function and dysfunction. Thromb Haemost 2001;86:138-43.
3. Akenami FO, Siren V, Koskiniemi M, et al. Cerebrospinal fluid activity of tissue plasminogen activator in patients with neurological diseases. J Clin Pathol 1996;49:577-80.
4. Philippe J, Offner F, Declerck PJ, et al. Fibrinolysis and coagulation in patients with infectious disease and sepsis. Thromb Haemost 1991;65:291-5.
5. Busch E, Kruger K, Fritze K, et al. Blood-brain barrier disturbances after rt-PA treatment of thromboembolic stroke in the rat. Acta Neurochir Suppl 1997;70:206-8.