Spontaneous venous pulsations should be monitored during glaucoma therapy [1]

British Journal of Ophthalmology

Volumn 86, Issue 10, 2002, Pages 1187-

  Parsa, C F ^a  

^a JOHNS HOPKINS UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CARBONATE DEHYDRATASE INHIBITOR;

CEREBROSPINAL FLUID PRESSURE; GLAUCOMA; HUMAN; INTRACRANIAL HYPERTENSION; INTRAOCULAR HYPERTENSION; LETTER; OPTIC NERVE DISEASE; PATHOGENESIS; PRIORITY JOURNAL;

GLAUCOMA; HUMANS; INTRACRANIAL PRESSURE; INTRAOCULAR PRESSURE; VENOUS PRESSURE;

EID: 0036784435     PISSN: 00071161     EISSN: None     Source Type: Journal    
DOI: 10.1136/bjo.86.10.1187     Document Type: Letter

References (6)

1. Volkov VV. Essential element of the glaucomatous process neglected in clinical practice [in Russian]. Oftalmol Zh 1976;31:500-4.
2. Morgan WH, Yu DY, Cooper RL, et al. The influence of cerebrospinal fluid pressure on the lamina cribrosa tissue gradient. Invest Ophthalmol Vis Sci 1995;36:1163-72.
3. Greenfield DS, Wanichwecharungruang B, Liebmann JM, et al. Pseudotumor cerebri appearing with unilateral papilledema after trabeculectomy. Arch Ophthalmol 1997;115:423-6.
4. Engel S. Venous pulsation as a symptom of early glaucoma. Am J Ophthalmol 1946;29:1446-8.
5. Rios-Montenegro EN, Anderson DR, David NJ. Intracranial pressure and ocular hemodynamics. Arch Ophthalmol 1973;89:52-8.
6. Firsching R, Schütze M, Motschmann M, et al. Venous ophthalmodynamometry: a noninvasive method for assessment of intracranial pressure. J Neurosurg 2000;93:33-6.