Cyclosporin A blocks muscle differentiation by inducing oxidative stress and inhibiting the peptidyl-prolyl-cis-trans isomerase activity of cyclophilin A: cyclophilin A protects myoblasts from cyclosporin A-induced cytotoxicity.

The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Volumn 16, Issue 12, 2002, Pages 1633-1635

  Hong, Feng ^a   Lee, Jinhwa ^a   Song, Jae Woo ^a   Lee, Su Jae ^a   Ahn, Heekyung ^a   Cho, Jeong Je ^a   Ha, Joohun ^a   Kim, Sung Soo ^a  

^a Kyung Hee University Hospital   (South Korea)

Author keywords

[No Author keywords available]

Indexed keywords

(MELLE 4)CYCLOSPORIN; (MELLE-4)CYCLOSPORIN; ASCOMYCIN; CALCINEURIN; CYCLOPHILIN A; CYCLOSPORIN; DRUG DERIVATIVE; IMMUNOSUPPRESSIVE AGENT; PEPTIDYLPROLYL ISOMERASE; REACTIVE OXYGEN METABOLITE; TACROLIMUS;

AMINO ACID SUBSTITUTION; ANIMAL; APOPTOSIS; ARTICLE; CELL DIFFERENTIATION; CELL LINE; CYTOLOGY; DRUG ANTAGONISM; DRUG EFFECT; DRUG RESISTANCE; GENETIC TRANSFECTION; GENETICS; HEART MUSCLE; METABOLISM; MUTATION; NICK END LABELING;

AMINO ACID SUBSTITUTION; ANIMALS; APOPTOSIS; CALCINEURIN; CELL DIFFERENTIATION; CELL LINE; CYCLOPHILIN A; CYCLOSPORINE; DRUG RESISTANCE; IMMUNOSUPPRESSIVE AGENTS; IN SITU NICK-END LABELING; MUTATION; MYOCARDIUM; PEPTIDYLPROLYL ISOMERASE; REACTIVE OXYGEN SPECIES; TACROLIMUS; TRANSFECTION;

EID: 0036778387     PISSN: None     EISSN: 15306860     Source Type: Journal    
DOI: 10.1096/fj.02-0060fje     Document Type: Article

References (0)