Mechanical stress-induced DNA damage and rac-p38MAPK signal pathways mediate p53-dependent apoptosis in vascular smooth muscle cells.

The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Volumn 16, Issue 11, 2002, Pages 1423-1425

  Mayr, Manuel ^a   Hu, Yanhua ^a   Hainaut, Hainaut ^a   Xu, Qingbo ^a  

^a ERICH SCHMID INSTITUTE OF MATERIALS SCIENCE   (Austria)

Author keywords

[No Author keywords available]

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE P38; PROTEIN BCL 2; PROTEIN P53; RAC PROTEIN;

ANIMAL; APOPTOSIS; ARTICLE; CELL CULTURE; CYTOLOGY; DNA DAMAGE; ENZYMOLOGY; MECHANICAL STRESS; METABOLISM; MITOCHONDRION; OXIDATIVE STRESS; PHYSIOLOGY; RAT; SIGNAL TRANSDUCTION; VASCULAR SMOOTH MUSCLE;

ANIMALS; APOPTOSIS; CELLS, CULTURED; DNA DAMAGE; MAP KINASE SIGNALING SYSTEM; MITOCHONDRIA; MITOGEN-ACTIVATED PROTEIN KINASES; MUSCLE, SMOOTH, VASCULAR; OXIDATIVE STRESS; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PROTO-ONCOGENE PROTEINS C-BCL-2; RAC GTP-BINDING PROTEINS; RATS; STRESS, MECHANICAL; TUMOR SUPPRESSOR PROTEIN P53;

MLCS; MLOWN;

EID: 0036719445     PISSN: None     EISSN: 15306860     Source Type: Journal    
DOI: 10.1096/fj.02-0042fje     Document Type: Article

References (0)