The N-ras proto-oncogene can suppress the malignant phenotype in the presence or absence of its oncogene

Cancer Research

Volumn 62, Issue 15, 2002, Pages 4514-4518

  Diaz, Roberto ^a   Ahn, Daniel ^a   Lopez Barcons, Lluis ^a,b   Malumbres, Marcos ^a,c   De Castro, Ignacio Perez ^a   Lue, Jeffrey ^a   Ferrer Miralles, Neus ^a,d   Mangues, Ramon ^a,e   Tsong, Jerry ^a   Garcia, Roberto ^a   Perez Soler, Roman ^a,b   Pellicer, Angel ^a  

^a NEW YORK UNIVERSITY MEDICAL CENTER   (United States)

^b ALBERT EINSTEIN COLLEGE OF MEDICINE   (United States)

^c CENTRO NACIONAL DE INVESTIGACIONES CARDIOVASCULARES CNIC   (Spain)

^d UNIVERSITAT POMPEU FABRA   (Spain)

^e HOSPITAL DE LA SANTA CREU I SANT PAU   (Spain)

Author keywords

[No Author keywords available]

Indexed keywords

3 METHYLCHOLANTHRENE; CARCINOGEN; GENOMIC DNA; METHYLNITROSOUREA;

ANIMAL CELL; ARTICLE; CELL GROWTH; GENE MUTATION; GENE OVEREXPRESSION; LYMPHOMA; MALIGNANT TRANSFORMATION; NONHUMAN; NUCLEOTIDE SEQUENCE; ONCOGENE N RAS; PHENOTYPE; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; PROTO ONCOGENE; THYMOMA; THYMUS LYMPHOMA; TRANSGENIC MOUSE;

ALLELES; ANIMALS; CROSSES, GENETIC; DNA MUTATIONAL ANALYSIS; FEMALE; GENES, RAS; GENETIC PREDISPOSITION TO DISEASE; LYMPHOMA; MALE; MICE; MICE, KNOCKOUT; RAS PROTEINS; THYMUS NEOPLASMS; TUMOR CELLS, CULTURED;

EID: 0036682167     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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