Computational biology of propagation in excitable media models of cardiac tissue

Chaos, Solitons and Fractals

Volumn 13, Issue 8, 2002, Pages 1643-1658

  Holden, A V ^a   Biktashev, V N ^b  

^a UNIVERSITY OF LEEDS   (United Kingdom)

^b UNIVERSITY OF LIVERPOOL   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ANISOTROPY; CARDIOLOGY; COMPUTATION THEORY; IONS; MATHEMATICAL MODELS; TISSUE;

CARDIAC GEOMETRY; COMPUTATIONAL BIOLOGY;

CELLS;

EID: 0036680905     PISSN: 09600779     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0960-0779(01)00172-2     Document Type: Conference Paper

References (46)

1. Modelling cardiac excitation and excitability
2. Re-entrant activity and its control in a model of mammalian ventricular tissue
3. Mathematical models of action potentials in the periphery and center of the rabbit sinoatrial node
4. Computational approaches to the ionic basis of pacemaker activity - From channel kinetics to the regional differences in rhythm
5. Mathematical model of the electrical activity of ventricular cell; Regional differences and propagation
6. Microscopic basis of anistropic propagation in the heart and the nature of current flow at a cellular level
7. Simulating cardiac sinus and atrial network dynamics on the connection machine
8. The effects of discrete gap junction coupling on propagation in myocardium
9. A mathematical model of cardiac anatomy
10. Re-entry in three-dimensional FitzHugh-Nagumo medium with rotational anisotropy
11. A mathematical model of cardiac anatomy
12. An algorithmic model of the Mammalian heart: Propagation, vulnerability, re-entry and fibrillation
13. Effects of shear flows on nonlinear waves in excitable media
14. Excitation wave breaking in excitable media with linear shear flow
15. The induction of reentry in cardiac tissue. The missing link: How electric fields alter transmembrane potential
16. The role of cardiac tissue structure in defibrillation
17. A generalized activating function for predicting virtual electrodes in cardiac tissue
18. A numerical method for the solution of bidomain equations in cardiac tissue
19. A model for the action of external current onto excitable tissue
20. Vulnerability in an excitable medium analytical and numerical studies of initiating unidirectional propagation
21. Preliminary report: Effect of encainide and flecainide on mortality in a randomised trial of arrhythmia suppression after myocardial infarction
22. Spiral wave meander and symmetry of the plane
23. On bifurcations of spiral waves in the plane
24. The restless heart of a spiral
25. Control of re-entrant activity in a model of mammalian atrial tissue
26. Characteristics of atrial re-entry and meander computed from a model of a single atrial cell
27. Self-terminating ventricular tachyarrhythmias - A diagnostic dilemma
28. Influence of the genotype on the clinical course of the long QT syndrome
29. Ion channels - Basic science and clinical disease
30. A plethora of mechanisms in the HERG related long QT syndrome - Genetics meets electrophysiology
31. Re-entrant arrhythmias in simulations of the Long-QT syndrome
32. Resonant drift of autowave vortices in two dimensions and the effects of boundaries and inhomogeneities
33. Design principles of a low voltage cardiac defibrillator based on the effect of feedback resonant drift
34. Fibrillation, chaos and clinical control
35. On feedback resonant drift and interaction with the boundaries in circular and annular excitable media
36. On the movement of excitation wavebreaks
37. Varieties of spiral wave behaviour: An experimentalist's approach to the theory of excitable media
38. Deterministic Brownian motion in the hypermeander of spiral waves
39. Three dimensional aspects of re-entry in experimental and numerical models of ventricular fibrillation
40. Tension of organizing filaments of scroll waves
41. Vortex dynamics in three-dimensional continuous myocardium with fiber rotation: Filament instability and fibrillation
42. Distribution of excitation frequencies on the epicardial and endocardial surfaces of fibrillating ventricular wall of the sheep heart
43. On two mechanisms of the domain structure of ventricular fibrillation
44. Intermittent conduction block can account for the spatial distribution of dominant frequencies during ventricular fibrillation