EB virus-encoded latent membrane protein 1 activates the JNK signalling pathway via a mechanism involving TRADD and TRAF in nasopharyngeal carcinoma cell

Progress in Biochemistry and Biophysics

Volumn 29, Issue 4, 2002, Pages 562-566

  Hu, Zhi ^a   Zeng, Liang ^a   Tao, Yong Guang ^a   Tang, Fa Qing ^a   Wang, Hai ^a   Luo, Fei Jun ^a   Yi, Wei ^a   Cao, Ya ^a  

^a Central South University   (China)

Author keywords

Activator protein 1; c Jun N terminal kinase; Epstein Barr virus (EB virus); Latent membrane protein 1; Nasopharyngeal carcinoma

Indexed keywords

LATENT MEMBRANE PROTEIN 1; STRESS ACTIVATED PROTEIN KINASE; TRANSCRIPTION FACTOR AP 1;

ARTICLE; CARBOXY TERMINAL SEQUENCE; CARCINOMA CELL; ENZYME ACTIVATION; EPSTEIN BARR VIRUS; MUTATION; NASOPHARYNX CARCINOMA; PROTEIN DOMAIN; PROTEIN EXPRESSION; PROTEIN INDUCTION; TIME; WESTERN BLOTTING;

EID: 0036676014     PISSN: 10003282     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (9)

1. Epstein-Barr virus latent membrane protein -1 (LMP1) signaling is distinct from CD40 and involves physical cooperation of its two C-terminus functional regions
2. NF-κB only partially mediates Epstein-Barr virus latent membrane protein 1 activation of B cells
3. Interaction of tumor necrosis factor receptor-associated factor signaling proteins with the LMP1 PXQXT motif is essential for induction of the EGFR expression
4. Tumor necrosis factor recepter-associated factor factor (TRAFs): A family of adaptor proteins that regulates life and death