Elevation of arterial potassium during acute systemic hypoxia is abolished by alkalosis

Journal of Cardiac Surgery

Volumn 17, Issue 4, 2001, Pages 342-346

  Mo, Fanny F M ^a   Essackjee, Hafejee C ^a   Ballard, Heather J ^a  

^a UNIVERSITY OF HONG KONG   (Hong Kong)

Author keywords

[No Author keywords available]

Indexed keywords

BICARBONATE; POTASSIUM;

ALKALOSIS; ANIMAL MODEL; ANIMAL TISSUE; ARTERIAL PH; CONFERENCE PAPER; CONTROLLED STUDY; DOG; ERYTHROCYTE; GRACILIS MUSCLE; HYPOXIA; MALE; NONHUMAN; PERFUSION PRESSURE; PERIPHERAL CIRCULATION; POTASSIUM TRANSPORT; RAT; VASODILATATION; VEIN BLOOD FLOW;

EID: 0036664989     PISSN: 08860440     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1540-8191.2001.tb01155.x     Document Type: Conference Paper

References (11)

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4. 2+] in oxygen-sensing tissues. Respir Physiol 1999;115:215-227.
5. Shigimori K, Ishizaki T, Matsukawa S, et al: Adenine nucleotides via activation of ATP-sensitive K+ channels modulate hypoxic response in rat pulmonary artery. Am J Physiol 1996;270:L803-809.
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10. Gibson JS, Ellory JC, Culliford SJ, et al: Volume-sensitive KCl cotransport and taurine fluxes in horse red blood cells. Exp Physiol 1993;78:685-695.
11. + efflux is highly expressed in young human red cells containing normal hemoglobin or HbS. J Membr Biol 1987;97:97-105.