Mice are a good model of human airway disease

American Journal of Respiratory and Critical Care Medicine

Volumn 166, Issue 1, 2002, Pages 5-6

  Gelfand, Erwin W ^a  

^a NATIONAL JEWISH MEDICAL AND RESEARCH CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANIMAL EXPERIMENT; ANIMAL MODEL; ASTHMA; CYTOKINE RELEASE; DISEASE MODEL; EDITORIAL; IMMUNE RESPONSE; MOUSE; NONHUMAN; PRIORITY JOURNAL; RESPIRATORY TRACT ALLERGY; RESPIRATORY TRACT DISEASE; SENSITIZATION; ANIMAL; HUMAN; IMMUNOLOGY; PATHOPHYSIOLOGY; SPECIES DIFFERENCE;

ANIMALS; ASTHMA; DISEASE MODELS, ANIMAL; HUMANS; MICE; RESPIRATORY HYPERSENSITIVITY; SPECIES SPECIFICITY;

EID: 0036644808     PISSN: 1073449X     EISSN: None     Source Type: Journal    
DOI: 10.1164/rccm.2204023     Document Type: Editorial

References (19)

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3. The late, but not early, asthmatic response is dependent on IL-5 and correlates with eosinophil infilatration
4. Prolonged antigen exposure ameliorates airway inflammation but not remodeling in a mouse model of bronchial asthma
5. A role for cysteinyl leukotrienes in airway remodeling in a mouse asthma model
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14. Immunostimulatory DNA sequences inhibit allergic (eosinophilic) inflammation in mice
15. Therapeutic strategies for allergic diseases
16. Anti-IgE efficacy in murine asthma models is dependent on the method of allergen sensitization
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19. Genetic variability in pulmonary physiological, cellular, and antibody responses to antigen in mice