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Volumn 7, Issue , 2002, Pages
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Chronic alcohol intoxication primes Kupffer cells and endothelial cells for enhanced CC-chemokine production and concomitantly suppresses phagocytosis and chemotaxis.
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Author keywords
[No Author keywords available]
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Indexed keywords
ALANINE AMINOTRANSFERASE;
BETA CHEMOKINE;
IMMUNOGLOBULIN ENHANCER BINDING PROTEIN;
MESSENGER RNA;
MNP 1 PROTEIN, HUMAN;
MNP-1 PROTEIN, HUMAN;
NUCLEAR PROTEIN;
SUPEROXIDE;
TRANSCRIPTION FACTOR;
TRANSCRIPTION FACTOR AP 1;
ALCOHOL INTOXICATION;
ALCOHOL LIVER DISEASE;
ANIMAL;
ARTICLE;
BACTERIUM ADHERENCE;
BIOSYNTHESIS;
BLOOD;
CHEMOTAXIS;
CHRONIC DISEASE;
CYTOLOGY;
DRUG EFFECT;
ENDOTHELIUM;
ESCHERICHIA COLI;
KUPFFER CELL;
LEUKOCYTE;
LIVER;
MALE;
METABOLISM;
PATHOLOGY;
PATHOPHYSIOLOGY;
PHAGOCYTOSIS;
PHYSIOLOGY;
RAT;
WISTAR RAT;
ALANINE TRANSAMINASE;
ALCOHOLIC INTOXICATION;
ANIMALS;
BACTERIAL ADHESION;
CHEMOKINES, CC;
CHEMOTAXIS;
CHRONIC DISEASE;
ENDOTHELIUM;
ESCHERICHIA COLI;
HEPATITIS, ALCOHOLIC;
KUPFFER CELLS;
LEUKOCYTES;
LIVER;
MALE;
NF-KAPPA B;
NUCLEAR PROTEINS;
PHAGOCYTOSIS;
RATS;
RATS, WISTAR;
RNA, MESSENGER;
SUPEROXIDES;
TRANSCRIPTION FACTOR AP-1;
TRANSCRIPTION FACTORS;
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EID: 0036617747
PISSN: None
EISSN: 10934715
Source Type: Journal
DOI: 10.2741/A746 Document Type: Article |
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Times cited : (46)
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References (0)
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