Physiological and pathological patterns in subthalamic neurons related to Parkison's disease

Neurophysiology

Volumn 34, Issue 2-3, 2002, Pages 96-

  Hammond, C ^a   Beurrier, C ^b   Garcia, L ^b   Bioulac, B ^b  

^a Turing Center for Living Systems CENTURI Aix Marseille University   (France)

^b CNRS   (France)

Author keywords

activity; High frequency stimulation; Parkinson's diasese; Subthalamic nucleus

Indexed keywords

4 AMINOBUTYRIC ACID RECEPTOR; CALCIUM; DOPAMINE; IONOTROPIC RECEPTOR; RESERPINE; SODIUM;

ANIMAL CELL; ANIMAL TISSUE; BRAIN SLICE; CALCIUM CURRENT; CONFERENCE PAPER; EVOKED RESPONSE; HYPERPOLARIZATION; IN VITRO STUDY; INTRACELLULAR RECORDING; ION CURRENT; NERVE CELL; NERVE CELL MEMBRANE POTENTIAL; NERVE CONDUCTION; NERVE STIMULATION; NONHUMAN; PARKINSON DISEASE; PATHOLOGY; PHYSIOLOGY; RAT; SODIUM CURRENT; SUBTHALAMIC NUCLEUS;

EID: 0036524213     PISSN: 00902977     EISSN: None     Source Type: Journal    
DOI: 10.1023/A:1020773015553     Document Type: Conference Paper

References (3)

1. NaP) underlies single-spike activity in rat subthalamic neurons," J. Neurophysiol., 83, 1951-1957 (2000).
2. C. Beurrier, P. Congar, B. Bioulac, and C. Hammond, "Subthalamic nucleus neurons switch from single-spike activity to burst-firing mode," J. Neurosci., 19, No. 2, 599-609 (1999).
3. C. Beurrier, B. Bioulac, J. Audin, and C. Hammond, "High-frequency stimulation produces a transient blockade of voltage-gated currents in subthalamic neurons," J. Neurophysiol., 85, 1351-1356 (2001).