Pathogenesis and treatment of renal dysfunction in rhabdomyolysis [3]

Intensive Care Medicine

Volumn 28, Issue 8, 2002, Pages 1185-

  Korantzopoulos, Panagiotis ^a   Galaris, Dimitrios ^a   Papaioannides, Dimitrios ^a  

^a UNIVERSITY OF IOANNINA   (Greece)

Author keywords

[No Author keywords available]

Indexed keywords

ASCORBIC ACID; MYOGLOBIN;

ALKALINIZATION; ANTIOXIDANT ACTIVITY; CHELATION; KIDNEY DYSFUNCTION; LETTER; LIPID PEROXIDATION; MOLECULAR MECHANICS; OXIDATION REDUCTION REACTION; OXIDATIVE STRESS; PATHOPHYSIOLOGY; RHABDOMYOLYSIS;

ANTIOXIDANTS; ASCORBIC ACID; HUMANS; HYDROGEN-ION CONCENTRATION; KIDNEY FAILURE, ACUTE; MYOGLOBIN; OXIDATION-REDUCTION; RHABDOMYOLYSIS; URINALYSIS;

EID: 0036360934     PISSN: 03424642     EISSN: None     Source Type: Journal    
DOI: 10.1007/s00134-002-1356-9     Document Type: Letter

References (5)

1. Pathogenesis and treatment of renal dysfunction in rhabdomyolysis
2. Redox cycling of myoglobin and ascorbate: A potential protective mechanism against oxidative reperfusion injury in muscle
3. On the molecular mechanism of metmyoglobin-catalyzed reduction of hydrogen peroxide by ascorbate
4. Ascorbic acid (vitamin C)
5. Vitamin C suppresses oxidative lipid damage in vivo, even in the presence of iron overload