Fra-1 substitutes for c-Fos in AP-1-mediated signal transduction in retinal apoptosis

Journal of Neurochemistry

Volumn 80, Issue 6, 2002, Pages 1089-1094

  Wenzel, Andreas ^a,c   Iseli, Hans Peter ^a   Fleischmann, Alexander ^b   Hafezi, Farhad ^a   Grimm, Christian ^a   Wagner, Erwin F ^b   Remé, Charlotte E ^a  

^a UNIVERSITY HOSPITAL ZURICH   (Switzerland)

^b RESEARCH INSTITUTE OF MOLECULAR PATHOLOGY   (Austria)

^c H Lab 13   (Switzerland)

Author keywords

Apoptosis; c Fos; Fra 1; Glucocorticoid receptor; Photoreceptor; Retina

Indexed keywords

GLUCOCORTICOID RECEPTOR; PROTEIN C FOS; TRANSCRIPTION FACTOR AP 1; TRANSCRIPTION FACTOR FRA 1;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; GENE REPRESSION; GENE TARGETING; GENOTYPE; LIGHT DAMAGE; MOUSE; NONHUMAN; PHOTORECEPTOR; PRIORITY JOURNAL; RETINA DEGENERATION; SIGNAL TRANSDUCTION;

ANIMALS; APOPTOSIS; DEXAMETHASONE; DISEASE MODELS, ANIMAL; GLUCOCORTICOIDS; IN SITU NICK-END LABELING; LIGHT; MICE; MICE, KNOCKOUT; PROTO-ONCOGENE PROTEINS C-FOS; RECEPTORS, GLUCOCORTICOID; RETINA; RETINAL DEGENERATION; SIGNAL TRANSDUCTION; TRANS-ACTIVATION (GENETICS); TRANSCRIPTION FACTOR AP-1;

ANIMALIA;

EID: 0036326048     PISSN: 00223042     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.0022-3042.2002.00807.x     Document Type: Article

References (23)

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