Requirements for the localization of p130 cas to Z-lines in cardiac myocytes

Cellular and Molecular Biology Letters

Volumn 7, Issue 2, 2002, Pages 323-329

  Kovacic Milivojevic, Branka ^a   Damsky, Caroline C ^a   Gardner, David G ^a   Ilić, Duško ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Cardiac Myocyte Hypertrophy; Cas; FAK; Z Lines

Indexed keywords

FOCAL ADHESION KINASE; PAXILLIN; PROTEIN P130; PROTEIN TYROSINE KINASE;

ANIMAL CELL; ANIMAL MODEL; CARBOXY TERMINAL SEQUENCE; CELL CULTURE; CELL DIFFERENTIATION; CONFERENCE PAPER; CONTROLLED STUDY; GENE EXPRESSION; HEART HEMODYNAMICS; HEART MUSCLE CELL; HEART VENTRICLE HYPERTROPHY; HEART WORK; HEMODYNAMIC MONITORING; IMMUNOCYTOCHEMISTRY; NEWBORN; NONHUMAN; PROTEIN INTERACTION; PROTEIN LOCALIZATION; PROTEIN MOTIF; RAT; SARCOMERE; SIGNAL TRANSDUCTION;

ANIMALIA; VERTEBRATA;

EID: 0036300094     PISSN: 14258153     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Conference Paper

References (4)

1. Focal adhesion kinase and p13 0Cas mediate both sarcomeric organization and activation of genes associated with cardiac myocyte hypertrophy
2. Requirements for localization of p130cas to focal adhesions
3. Signaling through focal adhesion kinase
4. Cardiovascular anomaly, impaired actin bundling and resistance to Src-induced transformation in mice lacking p130Cas