Understanding copper uptake at the molecular level

Nutrition Reviews

Volumn 60, Issue 6, 2002, Pages 177-179

  Wessling Resnick, Marianne ^a  

^a HARVARD SCHOOL OF PUBLIC HEALTH   (United States)

Author keywords

Copper efflux mechanisms; Ctr1; Cu ATPase genes; Menkes and Wilson diseases

Indexed keywords

ADENOSINE TRIPHOSPHATASE; COPPER; TRACE ELEMENT;

GENE MUTATION; MAMMAL CELL; MENKES SYNDROME; MOUSE; NONHUMAN; SHORT SURVEY; WILSON DISEASE;

ANIMALS; BIOLOGICAL TRANSPORT; CATION TRANSPORT PROTEINS; COPPER; HUMANS; MEMBRANE PROTEINS; MICE; MODELS, MOLECULAR;

MAMMALIA;

EID: 0036277755     PISSN: 00296643     EISSN: None     Source Type: Journal    
DOI: 10.1301/002966402320243269     Document Type: Short Survey

References (13)

1. Copper homeostasis: The role of cellular transporters
2. hCTR1: A human gene for copper uptake identified by complementation in yeast
3. Isolation of a murine copper transporter gene, tissue specific expression and functional complementation of a yeast copper transport mutant
4. The copper transporter CTR1 provides an essential function in mammalian embryonic development
5. Essential role for mammalian copper transporter Ctrl in copper homeostasis and embryonic development
6. Biochemical characterization of the human copper transporter Ctrl
7. Cloning and characterization of a mammalian protoncoupled metal-ion transporter
8. Microcytic anemia mice have a mutation in Nramp2, a candidate iron transporter gene
9. Characterization of the hCTR1 gene: Genomic organization, functional expression, and identification of a highly homologous processed gene
10. Separate pathways for cellular uptake of ferric and ferrous iron
11. Copper transport and metabolism are normal in aceruloplasminemic mice
12. Metallochaperones, an intracellular shuttle service for metal ions
13. The metallochaperone Atox1 plays a critical role in perinatal copper homeostasis