A new model for analyzing immunomodulation of T cell responses induced by oral administration of islet cell antigens

Annals of the New York Academy of Sciences

Volumn 958, Issue , 2002, Pages 179-181

  Bregenholt, Søren ^a   Wang, Mikala ^a   Zdravkovic, Maja ^a   Dyrberg, Thomas ^a   Petersen, Jacob S ^a  

^a NOVO NORDISK A S   (Denmark)

Author keywords

CD4+T cells; Insulin; Oral tolerance; Suppression; Type 1 diabetes

Indexed keywords

CELL ANTIGEN; GLUTAMATE DECARBOXYLASE; INSULIN; OVALBUMIN;

ANIMAL EXPERIMENT; AUTOIMMUNE DISEASE; CONFERENCE PAPER; CONTROLLED STUDY; DIABETES MELLITUS; DRUG TOLERANCE; FEMALE; IMMUNE RESPONSE; IMMUNIZATION; IMMUNOMODULATION; MOUSE; NONHUMAN; PANCREAS ISLET CELL; T LYMPHOCYTE;

ANIMALIA;

EID: 0036275517     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1111/j.1749-6632.2002.tb02965.x     Document Type: Conference Paper

References (8)

1. Oral tolerance in the treatment of inflammatory autoimmune diseases
2. Oral tolerance in disease
3. A cholera toxoid-insulin conjugate as an oral vaccine against spontaneous autoimmune diabetes
4. Suppression of insulitis in nonobese diabetic (NOD) mice by oral insulin administration is associated with selective expression of interleukin-4 and -10, transforming growth factor-b, and prostaglandin-E
5. Autoreactive CD4+ T cells protect from autoimmune diabetes via bystander supression using the IL-4/Stat6 pathway
6. Suppression of diabetes in nonobese diabetic mice by oral administartion of porcine insulin
7. Ligation of MHC class I molecules on peripheral blood T lymphocytes induces new phenotypes and functions
8. Future posibilities for prevention of type 1 diabetes