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Volumn 27, Issue 4, 2002, Pages 331-336
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Chronic cerebral hypoperfusion induces striatal alterations due to the transient increase of NO production and the depression of glutathione content
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Author keywords
Catalase; Glutathione; Lipid peroxidation; Nitric oxide; Oxidative stress; SOD
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Indexed keywords
CATALASE;
GLUTATHIONE;
NITRATE;
NITRIC OXIDE;
NITRITE;
SUPEROXIDE DISMUTASE;
THIOBARBITURIC ACID REACTIVE SUBSTANCE;
ANIMAL EXPERIMENT;
ANIMAL TISSUE;
ARTICLE;
BRAIN LEVEL;
BRAIN PERFUSION;
CONTROLLED STUDY;
CORPUS STRIATUM;
ENZYME ACTIVITY;
ENZYME SYNTHESIS;
LIPID PEROXIDATION;
NONHUMAN;
OXIDATION REDUCTION REACTION;
OXIDATIVE STRESS;
PRIORITY JOURNAL;
RECEPTOR BINDING;
ANIMALS;
CORPUS STRIATUM;
GLUTATHIONE;
KINETICS;
NITRATES;
NITRIC OXIDE;
NITRITES;
OXIDATIVE STRESS;
PERFUSION;
RATS;
ANIMALIA;
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EID: 0036209235
PISSN: 03643190
EISSN: None
Source Type: Journal
DOI: 10.1023/A:1014967414468 Document Type: Article |
Times cited : (19)
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References (33)
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