Protective effects of protein kinase C during myocardial ischemia require activation of phosphatidyl-inositol specific phospholipase C: Invited commentary

Annals of Thoracic Surgery

Volumn 73, Issue 4, 2002, Pages 1245-

  Matsuda, Hikaru ^a  

^a OSAKA UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

1 [[6 (3 METHOXYESTRA 1,3,5(10) TRIEN 17BETA YL)AMINO]HEXYL] 1H PYRROLE 2,5 DIONE; CALCIUM CHANNEL L TYPE; CHELERYTHRINE; PHORBOL 13 ACETATE 12 MYRISTATE; PHOSPHATIDYLINOSITOL; PHOSPHOLIPASE C; PHOSPHOLIPASE C INHIBITOR; PROTEIN KINASE C; PROTEIN KINASE C INHIBITOR;

ANIMAL TISSUE; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME INHIBITION; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HEART PROTECTION; ISOLATED HEART; NONHUMAN; NOTE; PRIORITY JOURNAL; RABBIT;

EID: 0036205513     PISSN: 00034975     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0003-4975(02)03483-5     Document Type: Note

References (3)

1. Ischemic preconditioning slows energy metabolism and delays ultrastructural damage during a sustained ischemic episode
2. Signal transduction by the phosphatidylinositol cycle in myocardium
3. P2Y purinoceptor activation mobilizes intracellular Ca2+ and induces a membrane current in rat intracardiac neurones