Failure of sensory neurons to express class I MHC is due to differential SOCS1 expression

Journal of Neuroimmunology

Volumn 123, Issue 1-2, 2002, Pages 35-40

  Turnley, Ann M ^a,b   Starr, Robyn ^a   Bartlett, Perry F ^a  

^a ROYAL MELBOURNE HOSPITAL   (Australia)

^b UNIVERSITY OF MELBOURNE   (Australia)

Author keywords

Cytokines; Histocompatibility; Interferon; Nervous system; Signal transduction

Indexed keywords

GAMMA INTERFERON; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 1; SUPPRESSOR FACTOR; SUPPRESSOR OF CYTOKINE SIGNALING 1; UNCLASSIFIED DRUG;

ANIMAL CELL; ANTIGEN EXPRESSION; ARTICLE; BRAIN INJURY; CELL SURVIVAL; CONTROLLED STUDY; GLIA; GLIA CELL; MALE; MOUSE STRAIN; NEWBORN; NONHUMAN; PRIORITY JOURNAL; PROTEIN INDUCTION; REGULATORY MECHANISM; SENSORY NERVE CELL; SIGNAL TRANSDUCTION; VIRUS INFECTION;

ANIMALS; CARRIER PROTEINS; CELLS, CULTURED; HISTOCOMPATIBILITY ANTIGENS CLASS I; INTERFERON TYPE II; MICE; MICE, INBRED C57BL; NEURONS, AFFERENT; REPRESSOR PROTEINS; SUPPRESSOR OF CYTOKINE SIGNALING PROTEINS;

EID: 0036187379     PISSN: 01655728     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0165-5728(01)00480-5     Document Type: Article

References (28)

1. Involvement of leukemia inhibitory factor and nerve growth factor in the development of dorsal root ganglion neurons
2. Upregulation of class I major histocompatibility complex gene expression in primary sensory neurons, satellite cells, and Schwann cells of mice in response to acute but not latent herpes simplex virus infection in vivo
3. Cell surface expression of H2 antigens on primary sensory neurons in response to acute but not latent herpes simplex virus infection in vivo