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Volumn 8, Issue 1, 2002, Pages 58-63
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Lack of nitric oxide synthase type 2 (NOS2) results in reduced neuronal apoptosis and mortality following mouse hepatitis virus infection of the central nervous system
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Author keywords
Apoptosis; Demyelination; Neuroimmunology; Nitric oxide; Virus
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Indexed keywords
NITRIC OXIDE;
NITRIC OXIDE SYNTHASE;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
APOPTOSIS;
ARTICLE;
BRAIN LEVEL;
BRAIN NERVE CELL;
CENTRAL NERVOUS SYSTEM INFECTION;
CLEARANCE;
CONFOCAL MICROSCOPY;
CONTROLLED STUDY;
CORRELATION FUNCTION;
DEMYELINATION;
MORTALITY;
MOUSE;
MURINE HEPATITIS CORONAVIRUS;
NICK END LABELING;
NONHUMAN;
PRIORITY JOURNAL;
VIRUS PATHOGENESIS;
ANIMALS;
APOPTOSIS;
BRAIN;
CORONAVIRUS INFECTIONS;
DEMYELINATING DISEASES;
ENCEPHALITIS, VIRAL;
IN SITU NICK-END LABELING;
MICE;
MICE, INBRED C57BL;
MICE, KNOCKOUT;
MURINE HEPATITIS VIRUS;
NEURONS;
NITRIC OXIDE SYNTHASE;
NITRIC OXIDE SYNTHASE TYPE II;
ANIMALIA;
CORONAVIRUS;
MURINAE;
MURINE HEPATITIS VIRUS;
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EID: 0036185507
PISSN: 13550284
EISSN: None
Source Type: Journal
DOI: 10.1080/135502802317247820 Document Type: Article |
Times cited : (16)
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References (37)
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