Apoptotic effect of tetra-arsenic tetra-sulfide on acute promyelocytic leukemia cell line NB4

Chinese Journal of Pharmacology and Toxicology

Volumn 16, Issue 1, 2002, Pages 37-40

  Hao, Hong Ying ^a   Teng, Zhi Ping ^a   Lu, Dao Pei ^a  

^a PEKING UNIVERSITY   (China)

Author keywords

Cell, NB4; Genes; Leukemia, promyelocytic, acute; Receptor, retinoic acid; Tetra arsenic tetra sulfide

Indexed keywords

ANTILEUKEMIC AGENT; ARSENIC DERIVATIVE; HYBRID PROTEIN; RETINOIC ACID RECEPTOR ALPHA; TETRAARSENIC TETRASULFIDE; UNCLASSIFIED DRUG;

APOPTOSIS; ARTICLE; CELL STRUCTURE; DRUG EFFECT; DRUG MECHANISM; FLOW CYTOMETRY; FLUORESCENCE IN SITU HYBRIDIZATION; FUSION GENE; GENE EXPRESSION; HUMAN; HUMAN CELL; PROMYELOCYTIC LEUKEMIA; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; WESTERN BLOTTING;

EID: 0036183136     PISSN: 10003002     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (9)

1. Arsenicals in hematologic cancers
2. 4), a monoinstitutional study
3. A rapid and simple method for the isolation of apoptotic DNA fragment
4. Molecular monitoring of the myl/retinoic acid receptor-alpha fusion gene in acute promyelocytic leukemia by polymerase chain reaction
5. Control of programmed cell death in normal and leukemic cells: New implications for therapy
6. Apoptosis in pathogenesis and treatment of disease
7. The t(15; 17) translocation of acute promyelocytic leukemia fuses the retinoic acid receptor alpha gene to a novel transcribed locus