Inflammation in acute coronary syndromes

Heart

Volumn 87, Issue 3, 2002, Pages 201-204

  Mulvihill, N T ^a   Foley, J B ^a  

^a ST JAMES S HOSPITAL   (Ireland)

Author keywords

[No Author keywords available]

Indexed keywords

BIOLOGICAL MARKER; C REACTIVE PROTEIN; CYTOKINE; ENDOTHELIN; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERCELLULAR ADHESION MOLECULE 1; VASCULAR CELL ADHESION MOLECULE 1; VASOCONSTRICTOR AGENT;

ATHEROSCLEROTIC PLAQUE; BLOOD VESSEL REACTIVITY; CHLAMYDOPHILA PNEUMONIAE; CORONARY ARTERY ATHEROSCLEROSIS; CORONARY ARTERY DISEASE; CYTOMEGALOVIRUS; DISEASE ASSOCIATION; HEART MUSCLE ISCHEMIA; HUMAN; INFECTION; INFLAMMATION; MOLECULAR MECHANICS; PATHOGENESIS; PRIORITY JOURNAL; REVIEW;

EID: 0036181394     PISSN: 13556037     EISSN: None     Source Type: Journal    
DOI: 10.1136/heart.87.3.201     Document Type: Review

References (47)

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3. Pathophysiology of acute coronary syndromes
4. Significance of adventitial inflammation of the coronary artery in patients with unstable angina: Results of autopsy
5. Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterised by an inflammatory process irrespective of the dominant plaque morphology
6. Accumulation of activated mast cells in the shoulder regions of human coronary atheroma, the predilection site of atheromatous rupture
7. Association between myocardial infarction and the mast cells in the adventitia of infarct-related coronary artery
8. Recent activation of the plaque immune response in coronary lesions underlying acute coronary syndromes
9. Acute T-cell activation is detectable in unstable angina
10. Apoptosis of vascular smooth muscle cells in vascular remodelling and atherosclerolic plaque rupture
11. Interstitial collagenase [MMP-1) expression in human carotid atherosclerosis
12. Mechanisms of disease: Nuclear factor-κB: A pivitol transcription factor in chronic inflammatory diseases
13. Nuclear factor-kB is selectively and markedly activated in humans with unstable angina pectoris
14. Increased expression of neutrophil and monocyte adhesion molecules in unstable coronary artery disease
15. Influx of neutrophils into the walls of large epicardial coronary arteries in response to ischaemia/reperfusion
16. Intercellular neutrophil myeloperoxidase is reduced in unstable angina and acute myocardial infarction, but its reduction is not related to ischaemia
17. Myocardial reperfusion injury: Experimental evidence and clinical relevance
18. Tissue factor generation by human mononuclear cells: Effects of endotoxin and dissociation of tissue factor generation from mitogenic response
19. A transient intermittent lymphocyte activation is responsible for instability in angina
20. Acute T-cell activation is detectable in unstable angina
21. Release of prostaglandins and thromboxane into the coronary circulation in patients with ischemic heart disease
22. Elevation of C-reactive protein in "active" coronary artery disease
23. Measurement of serum C-reactive protein concentration in myocardial ischaemia and infarction
24. The prognostic value of C-reactive protein and serum amyloid A protein in severe unstable angina
25. Increasing levels of interleukin (IL)-1 Ra and IL-6 during the first 2 days of hospitalisation in unstable angina are associated with an increased risk of in-hospital coronary events
26. Inflammation, aspirin and the risk of cardiovascular disease in apparently healthy men
27. Prospective study of C-reactive protein and the risks of future cardiovascular events among apparently healthy women
28. Regulation of complement activation by C-reactive protein
29. C-reactive protein frequently colocalizes with the terminal complement complex in the intima of early atherosclerotic lesions of human coronary arleries
30. C-reactive protein induces human peripheral blood monocytes to synthesize tissue factor
31. Expression of intercellular adhesion molecule-1 in atherosclerolic plaques
32. Direct proinflammatory effect of C-reactive protein on human endothelial cells
33. Circulating ICAM-1 and E-selectin in patients with acute coronary syndrome
34. Increased soluble form of P-selectin in patients with unstable angina
35. Early temporal expression of soluble cellular adhesion molecules in unstable angina and subendocardial myocardial infarction
36. Inflammatory markers predicting outcome in unstable angina and non Q-wave myocardial infarction
37. Unstable angina: Natural history and determinants of prognosis
38. Elevated levels of C-reactive protein of discharge in patients with unstable angina predict recurrent instability
39. Platelet activation in patients after an acute coronary syndrome: Results from the TIMI-12 trial. Thrombolysis in myocardial infarction
40. Evidence of prolonged inflammation in unstable angina and non Q-wave myocardia/infarction
41. Cytomegalovirus and atherosclerosis
42. Association between prior cytomegalovirus infection and the risk of restenosis after coronary atherectomy
43. Cytomegalovirus infection is associated with cardiac allograft rejection and atherosclerosis
44. Serological evidence of an association of a novel chlamydia, TWAR, with chronic coronary heart disease and acute myocardial infarction
45. Independent and joint effects of antibodies to human heat shock protein 60 and chlamydia pneumoniae infection in the development of coronary atherosclerosis
46. Increased incidence of chlamydia species within the coronary arteries of patients with symptomatic atherosderotic versus other forms of cardiovascular disease
47. High prevalence of latently present cytomegalovirus in arterial walls of patients suffering from grade III atherosclerosis