Immunoregulation during disease progression in prediabetic NOD mice: Inverse expression of arginase and prostaglandin H synthase 2 vs. Interleukin-15

Hormone and Metabolic Research

Volumn 34, Issue 1, 2002, Pages 7-12

  Rothe, H ^a,b   Hausman, A ^a   Kolb, H ^a  

^a HEINRICH HEINE UNIVERSITY   (Germany)

^b INSTITUT FÜR DIABETESFORSCHUNG   (Germany)

Author keywords

Cyclophosphamide; Cytokines; Innate Immunity; Insulitis; Macrophages; Non Obese Diabetic Mice

Indexed keywords

ARGINASE; CYCLOPHOSPHAMIDE; GAMMA INTERFERON; INTERLEUKIN 12; INTERLEUKIN 15; INTERLEUKIN 18; MESSENGER RNA; NITRIC OXIDE SYNTHASE INHIBITOR; PROSTAGLANDIN SYNTHASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL INVASION; CONTROLLED STUDY; CORRELATION FUNCTION; CYTOKINE PRODUCTION; CYTOTOXIC T LYMPHOCYTE; DIABETES MELLITUS; DISEASE ASSOCIATION; DISEASE COURSE; DOWN REGULATION; FEMALE; GENE EXPRESSION; IMMUNOCOMPETENT CELL; IMMUNOREGULATION; IMPAIRED GLUCOSE TOLERANCE; INSULIN DEPENDENT DIABETES MELLITUS; INSULITIS; MACROPHAGE; MOUSE; NONHUMAN; PANCREAS DISEASE; PRIORITY JOURNAL; PROTEIN EXPRESSION;

ANIMALS; ARGINASE; DIABETES MELLITUS, EXPERIMENTAL; FEMALE; GENE EXPRESSION REGULATION; HISTOCYTOCHEMISTRY; INTERLEUKIN-15; INTERLEUKINS; MICE; MICE, INBRED NOD; PANCREAS; PROSTAGLANDIN-ENDOPEROXIDE SYNTHASES; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; RNA, MESSENGER; STATISTICS, NONPARAMETRIC; UP-REGULATION;

ANIMALIA;

EID: 0036170643     PISSN: 00185043     EISSN: None     Source Type: Journal    
DOI: 10.1055/s-2002-19959     Document Type: Article

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