Allelic loss in clinically and screening-detected primary hyperparathyroidism

Clinical Endocrinology

Volumn 56, Issue 1, 2002, Pages 113-117

  Correa, Pamela ^a   Juhlin, Christofer ^a   Rastad, Jonas ^a,b   Åkerström, Göran ^a   Westin, Gunnar ^a   Carling, Tobias ^a,c  

^a UPPSALA UNIVERSITY HOSPITAL   (Sweden)

^b ASTRAZENECA R AND D   (Sweden)

^c BURNHAM INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM; FLUORESCENT DYE; PARATHYROID HORMONE;

ADULT; AGED; ALLELE; ARTICLE; CALCIUM BLOOD LEVEL; CHROMOSOME ANALYSIS; CLINICAL FEATURE; CLINICAL TRIAL; CONTROLLED CLINICAL TRIAL; CONTROLLED STUDY; DISEASE ASSOCIATION; FEMALE; HETEROZYGOSITY LOSS; HORMONE BLOOD LEVEL; HUMAN; HUMAN TISSUE; ISOTOPE LABELING; MAJOR CLINICAL STUDY; MALE; MEDICAL PRACTICE; MICROSATELLITE MARKER; ORGAN WEIGHT; PARATHYROID TUMOR; PRIMARY HYPERPARATHYROIDISM; PRIORITY JOURNAL; SCREENING; TUMOR SUPPRESSOR GENE;

AGED; CHI-SQUARE DISTRIBUTION; CHROMOSOMES, HUMAN, PAIR 1; CHROMOSOMES, HUMAN, PAIR 11; CHROMOSOMES, HUMAN, PAIR 6; FEMALE; HUMANS; HYPERPARATHYROIDISM; LOSS OF HETEROZYGOSITY; MALE; MASS SCREENING; MIDDLE AGED;

EID: 0036153518     PISSN: 03000664     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.0300-0664.2001.01436.x     Document Type: Article

References (27)

1. Parathyroidectomy for primary hyperparathyroidism induces positive uncoupling and increases bone mineral density in cancellous bone
2. Differential genetic alterations in von Hippel-Lindau syndrome-associated and sporadic pheochromocytomas
3. Sporadic and familial pheochromocytomas are associated with loss of at least two discrete intervals on chromosome 1p
4. Molecular pathology of parathyroid tumours
5. Parathyroid MEN1 gene mutations in relation to clinical characteristics of non-familial primary hyperparathyroidism
6. Analysis of the RAD54 gene on chromosome 1p as a potential tumour-suppressor gene in parathyroid adenomas
7. Primary hyperparathyroidism: Effect of parathyroidectomy on regional bone mineral density in Danish patients. A 3-year follow-up study
8. Frequent loss of chromosome arm 1p DNA in parathyroid adenomas
9. Allelotype of human breast carcinoma: A second major site for loss of heterozygosity is on chromosome 6q
10. Differential loss of heterozygosity in familial, sporadic, and uremic hyperparathyroidism
11. Alterations of the MEN1 gene in sporadic parathyroid tumours
12. Allelic loss from chromosome 11 in parathyroid tumours
13. Somatic mutations of the MEN1 gene in parathyroid tumours
14. Genetic alterations on 3p, 11q13, and 18q in nonfamilial and MEN1-associated pancreatic endocrine tumours
15. Cyclin D1/PRAD1 expression in parathyroid adenomas: An immunohistochemical study
16. Loss of heterozygosity suggests multiple genetic alterations in pheochromocytomas and medullary thyroid carcinomas
17. A cure in search of a disease: Parathyroidectomy for non-traditional features of primary hyperparathyroidism
18. Population-based health screening for primary hyperparathyroidism with serum calcium and parathyroid hormone values in menopausal women
19. Epidemiology of primary hyperparathyroidism
20. Prediction of bone mass change after parathyroidectomy in patients with primary hyperparathyroidism
21. Novel chromosomal abnormalities identified by comparative genomic hybridisation in parathyroid adenomas
22. Prevalence of hypercalcemia in a health survey: A 14-year follow-up study of serum calcium values
23. Profile of clinical practice: Thresholds for surgery and surgical outcomes for patients with primary hyperparathyroidism. A national survey of endocrine surgeons
24. Genomic localisation of novel candidate tumour suppressor gene loci in human parathyroid adenomas
25. Parathyroid tumour suppressor on 1p: Analysis of the p18 cyclin-dependent kinase inhibitor gene as a candidate
26. Localisation of a gene causing endocrine neoplasia to a 4 cm region on chromosome
27. Primary hyperparathyroidism