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Volumn 14, Issue 3, 2002, Pages 211-218
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Redox regulation of TNF-α biosynthesis: Augmentation by irreversible inhibition of γ-glutamylcysteine synthetase and the involvement of an IκB-α/NF-κB-independent pathway in alveolar epithelial cells
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Author keywords
Antioxidant; Chemioxyexcitation; Cytokines; Glutathione disequilibrium; Pharmacotherapy; Reactive oxygen species; Redox thiol regulation
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Indexed keywords
BUTHIONINE SULFOXIMINE;
ENZYME INHIBITOR;
GLUTAMATE CYSTEINE LIGASE;
GLUTATHIONE;
I KAPPA B;
IMMUNOGLOBULIN ENHANCER BINDING PROTEIN;
LIPOPOLYSACCHARIDE;
OXIDIZING AGENT;
TUMOR NECROSIS FACTOR ALPHA;
ANIMAL CELL;
ARTICLE;
CONTROLLED STUDY;
CYTOKINE PRODUCTION;
CYTOSOL;
DOSE RESPONSE;
ENZYME INHIBITION;
FETUS;
LUNG ALVEOLUS EPITHELIUM;
MODEL;
NONHUMAN;
OXIDATION REDUCTION REACTION;
PRIORITY JOURNAL;
PROTEIN DEGRADATION;
PROTEIN PHOSPHORYLATION;
RAT;
REGULATORY MECHANISM;
SIGNAL PROCESSING;
SIGNAL TRANSDUCTION;
ANIMALS;
BUTHIONINE SULFOXIMINE;
CELLS, CULTURED;
DIPEPTIDES;
DNA-BINDING PROTEINS;
DOSE-RESPONSE RELATIONSHIP, DRUG;
ENZYME INHIBITORS;
EPITHELIAL CELLS;
FEMALE;
GLUTAMATE-CYSTEINE LIGASE;
I-KAPPA B PROTEINS;
LIPOPOLYSACCHARIDES;
LUNG;
NF-KAPPA B;
OXIDATION-REDUCTION;
RATS;
RATS, SPRAGUE-DAWLEY;
SIGNAL TRANSDUCTION;
TIME FACTORS;
TUMOR NECROSIS FACTOR-ALPHA;
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EID: 0036153161
PISSN: 08986568
EISSN: None
Source Type: Journal
DOI: 10.1016/S0898-6568(01)00233-9 Document Type: Article |
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Times cited : (37)
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References (35)
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